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Natsue Omi

Researcher at Kyoto Prefectural University of Medicine

Publications -  9
Citations -  346

Natsue Omi is an academic researcher from Kyoto Prefectural University of Medicine. The author has contributed to research in topics: Genome-wide association study & Single-nucleotide polymorphism. The author has an hindex of 6, co-authored 8 publications receiving 317 citations.

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Three susceptible loci associated with primary open-angle glaucoma identified by genome-wide association study in a Japanese population

TL;DR: It turned out that 3 genetic loci probably associated with POAG have been identified, and these findings would provide the foundation for future studies to build on, such as for the metaanalysis, to reveal the molecular mechanism of the POAG pathogenesis.
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Common Variants in CDKN2B-AS1 Associated with Optic-Nerve Vulnerability of Glaucoma Identified by Genome-Wide Association Studies in Japanese

TL;DR: POAG-associated variants in the CDKN2B-AS1 locus are identified using a Japanese population and could be associated specifically with the vulnerability of the optic nerve to IOP, which is useful for investigating the etiology of glaucoma.
Journal Article

LOXL1 genetic polymorphisms are associated with exfoliation glaucoma in the Japanese population

TL;DR: Although the functional effects of the LOXL1 SNP appear to be qualitative rather than quantitative, the amino acid substitution (R141L) caused by SNP rs1048661 is not a simple decisive factor for XFG due to the inverted allele frequency between Japanese XFG and Caucasian XFG patients.
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Novel common variants and susceptible haplotype for exfoliation glaucoma specific to Asian population

TL;DR: The results suggested that the combination of newly discovered variants in these genes might be useful for precise XFG risk assessment, as well as for elucidating the molecular mechanism of XFG pathogenesis through XFS.
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Syndactyly and preaxial synpolydactyly in the single Sfrp2 deleted mutant mice.

TL;DR: The results suggest that the Sfrp2 deleted mice can be a useful animal model to study human syndactyly/preaxial synpolydactYly defects.