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Neil Pearce

Researcher at University of London

Publications -  795
Citations -  122260

Neil Pearce is an academic researcher from University of London. The author has contributed to research in topics: Population & Asthma. The author has an hindex of 107, co-authored 729 publications receiving 105762 citations. Previous affiliations of Neil Pearce include Harvard University & Victoria University of Wellington.

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Transmission and Control of SARS-CoV-2 in the Food Production Sector: A Rapid Narrative Review of the Literature

TL;DR: Findings showed some evidence of a high transmission level of SARS-CoV-2 within some areas of the food production sector, and preventative measures included/recommended were social distancing, testing, adequate ventilation, cleaning regimes and access to PPE.

Transactional sex in the wake of COVID-19: sexual and reproductive health and rights of the forcibly displaced A Test-Negative Design with Additional Population Controls Can Be Used to Rapidly Study Causes of the SARS-CoV-2 Epidemic

TL;DR: In this article, two types of case-control studies that can be carried out jointly in test settings for symptomatic persons are proposed: test-negative case control design (TND) and standard case control studies with population controls.
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Prevalence of and Risk Factors for Chronic Kidney Disease of Unknown Aetiology in India

TL;DR: A large number of patients in central America and Sri Lanka have reported a high burden of chronic kidney disease of unknown aetiology (CKDu), and it is unknown whether CKDu occurs in other regions of the world.
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The influence of early-life animal exposure on the risk of childhood atopic dermatitis, asthma and allergic rhinoconjunctivitis: findings from the Danish National Birth Cohort.

TL;DR: In this paper , the authors explored factors that may modify associations of early-life animal exposure with asthma and allergic disease, so as to better understand these differences in findings, and suggested that these factors should be considered when assessing the risks associated with early life animal exposure, suggesting that the overall weak associations observed between animal exposure and atopic dermatitis, asthma, and allergic rhinoconjunctivitis were modified by type of animal, source of exposure, parental history of asthma or allergy and timing of exposure.