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Nobunao Wakabayashi

Researcher at University of Pittsburgh

Publications -  51
Citations -  18539

Nobunao Wakabayashi is an academic researcher from University of Pittsburgh. The author has contributed to research in topics: Transcription factor & Signal transduction. The author has an hindex of 36, co-authored 48 publications receiving 16561 citations. Previous affiliations of Nobunao Wakabayashi include Johns Hopkins University & Johns Hopkins University School of Medicine.

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Keap1 represses nuclear activation of antioxidant responsive elements by Nrf2 through binding to the amino-terminal Neh2 domain

TL;DR: It is postulate that Keap1 and Nrf2 constitute a crucial cellular sensor for oxidative stress, and together mediate a key step in the signaling pathway that leads to transcriptional activation by this novel NRF2 nuclear shuttling mechanism.
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Cell Survival Responses to Environmental Stresses Via the Keap1-Nrf2-ARE Pathway

TL;DR: The development of Nrf2 knockout mice has provided key insights into the toxicological importance of this pathway, and this review highlights the key elements in this adaptive response to protection against acute and chronic cell injury provoked by environmental stresses.
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Protection against electrophile and oxidant stress by induction of the phase 2 response: fate of cysteines of the Keap1 sensor modified by inducers.

TL;DR: Evidence for formation of intermolecular disulfide bridges was obtained by 2D PAGE of extracts of cells treated with inducers, and by the demonstration that whereas C273A and C288A mutants of Keap1 alone could not repress Nrf2 activation of the ARE-luciferase reporter, an equal mixture of these mutant constructs restored repressor activity.
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Keap1-null mutation leads to postnatal lethality due to constitutive Nrf2 activation

TL;DR: These experiments show that Keap1 acts upstream of Nrf2 in the cellular response to oxidative and xenobiotic stress, and breeding to NRF2-deficient mice reversed the phenotypic Keap 1 deficiencies.