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Noel N. Kim

Researcher at Boston University

Publications -  126
Citations -  6898

Noel N. Kim is an academic researcher from Boston University. The author has contributed to research in topics: Receptor & Vagina. The author has an hindex of 43, co-authored 116 publications receiving 6320 citations. Previous affiliations of Noel N. Kim include Boston Medical Center & University of Pennsylvania.

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A nitric oxide-like factor mediates nonadrenergic-noncholinergic neurogenic relaxation of penile corpus cavernosum smooth muscle.

TL;DR: It is concluded that endothelium-mediated responses of corpus cavernosum smooth muscle are mediated by a diffusible nitric oxide-like substance, and NANC neurogenic inhibitory responses do not require functional endot Helium, and nitricoxide, or a closely related substance, may act as an inhibitory neurotransmitter in penile corpus cavernsum smooth Muscle.
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Identification of functional angiotensin II receptors on rat cardiac fibroblasts.

TL;DR: Results from this study indicate that neonatal and adult rat cardiac fibroblasts in culture express AT, receptors for Ang II, and suggest that Ang II might have important regulatory roles over cardiac Fibroblast function under normal and pathological conditions.
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Anatomy, Physiology, and Pathophysiology of Erectile Dysfunction

TL;DR: Despite the efficacy of current therapies, they remain insufficient to address growing patient populations, such as those with diabetes and MetS, and increasing awareness of the adverse side effects of commonly prescribed medications on sexual function provides a rationale for developing new treatment strategies that minimize the likelihood of causing sexual dysfunction.
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Oxygen tension regulates the nitric oxide pathway. Physiological role in penile erection.

TL;DR: Investigation of the physiological role of oxygen tension in the regulation of trabecular smooth muscle tone concluded that physiological concentrations of oxygen modulate penile erection by regulating nitric oxide synthesis in corpus cavernosum tissue.
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Effects of castration and androgen replacement on erectile function in a rabbit model.

TL;DR: The results of this study demonstrate that androgen deprivation alters the functional responses and structure of erectile tissue.