N
Norio Ishida
Researcher at Tokyo Institute of Technology
Publications - 35
Citations - 1598
Norio Ishida is an academic researcher from Tokyo Institute of Technology. The author has contributed to research in topics: Circadian rhythm & Suprachiasmatic nucleus. The author has an hindex of 17, co-authored 35 publications receiving 1560 citations. Previous affiliations of Norio Ishida include Japanese Ministry of International Trade and Industry & National Institute of Advanced Industrial Science and Technology.
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Journal ArticleDOI
Multitissue Circadian Expression of Rat periodHomolog (rPer2) mRNA Is Governed by the Mammalian Circadian Clock, the Suprachiasmatic Nucleus in the Brain
Katsuhiko Sakamoto,Takahiro Nagase,Hiromi Fukui,Kazumasa Horikawa,Tetsuya Okada,Hitoshi Tanaka,Kazuo Sato,Yoshiaki Miyake,Osamu Ohara,Koichiro Kako,Norio Ishida +10 more
TL;DR: It is suggested that the multitissue circadian expression of rPer2 mRNA was governed by the mammalian brain clock SCN and also suggest that the r per2 gene was involved in the circadian rhythm of locomotor behavior in mammals.
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Antiphase circadian expression between BMAL1 and period homologue mRNA in the suprachiasmatic nucleus and peripheral tissues of rats.
TL;DR: The amplitudes of BMAL1 and rPer2 mRNA expression levels were correlated between the different tissues, suggesting that the circadian expression of BMal1 mRNA plays an important role in generating the circadianexpression of per homologue genes in mammals.
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Rhythmic expression of BMAL1 mRNA is altered in Clock mutant mice: differential regulation in the suprachiasmatic nucleus and peripheral tissues.
TL;DR: Observations suggest that the circadian expression of BMAL1 mRNA is affected by the CLOCK-induced transcriptional feedback loop in the SCN and peripheral tissues in a different way and that the regulation mechanism appeared to be different from those in mPer2 and DBP expressions in vivo.
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Humoral signals mediate the circadian expression of rat period homologue (rPer2) mRNA in peripheral tissues
TL;DR: Northern blot analysis revealed the circadian expression of rat period homologue (rPer2) mRNA in peripheral mononuclear leukocytes that have no neuronal connections, suggesting that some humoral signals, driven by the SCN, mediate the circadianexpression of mammalian per homologues in peripheral tissues.
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Functional CLOCK is not involved in the entrainment of peripheral clocks to the restricted feeding: entrainable expression of mPer2 and BMAL1 mRNAs in the heart of Clock mutant mice on Jcl:ICR background.
TL;DR: The restricted feeding (RF) shifted the circadian phase of both mPer2 and BMAL1 mRNA expressions in the heart not only of wild-type mice but also of Clock mutant mice, indicating that functional CLOCK is not required for an entrainment of peripheral clocks to RF.