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Olwenn V. Martin

Researcher at Brunel University London

Publications -  46
Citations -  1728

Olwenn V. Martin is an academic researcher from Brunel University London. The author has contributed to research in topics: Medicine & Environmental exposure. The author has an hindex of 19, co-authored 38 publications receiving 1067 citations. Previous affiliations of Olwenn V. Martin include Imperial College London & University College London.

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Effects of music in exercise and sport: A meta-analytic review.

TL;DR: Overall, results supported the use of music listening across a range of physical activities to promote more positive affective valence, enhance physical performance, reduce perceived exertion, and improve physiological efficiency.
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Ten years of research on synergisms and antagonisms in chemical mixtures: A systematic review and quantitative reappraisal of mixture studies.

TL;DR: A systematic review and quantitative reappraisal of 10 years' of experimental mixture studies to investigate the frequency and reliability of evaluations of mixture effects as synergistic or antagonistic confirmed previous concerns about the synergistic potential of combinations of triazine, azole and pyrethroid pesticides at environmentally relevant doses.
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Scientific Challenges in the Risk Assessment of Food Contact Materials

TL;DR: Current regulations are insufficient for addressing chemical exposures from FCAs, and exposures to hazardous substances migrating from FC as well as developing novel approaches for assessing the safety of chemicals in FCAs are recommended.

State of the art assessment of endocrine disruptors: Final Report

TL;DR: The Japanese translation lies entirely with the Division of Cellular and Molecular Toxicology, Biological Safety Research Center, National Institute of Health Sciences.
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Testicular Dysgenesis Syndrome and the Estrogen Hypothesis: A Quantitative Meta-Analysis

TL;DR: A quantitative meta-analysis of the association between the end points related to TDS and prenatal exposure to estrogenic agents found the doubling of the risk ratios for all three end points investigated after DES exposure is consistent with a shared etiology and the TDS hypothesis but does not constitute evidence of an estrogenic mode of action.