O
Osamu Matsubara
Researcher at National Defense Medical College
Publications - 136
Citations - 9108
Osamu Matsubara is an academic researcher from National Defense Medical College. The author has contributed to research in topics: Carcinoma & Adenocarcinoma. The author has an hindex of 42, co-authored 135 publications receiving 8511 citations. Previous affiliations of Osamu Matsubara include Vanderbilt University & Japanese Foundation for Cancer Research.
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Journal ArticleDOI
American thoracic society/European respiratory society international multidisciplinary consensus classification of the idiopathic interstitial pneumonias
William D. Travis,Talmadge E. King,Eric D. Bateman,David A. Lynch,Frédrique Capron,Thomas V. Colby,Jean-François Cordier,Roland M. Dubois,Jeffrey R. Galvin,Philippe Grenier,David M. Hansell,Gary W. Hunninghake,Masanori Kitaichi,Nestor L. Müller,Jeffrey L. Myers,Sonoko Nagai,Andrew G. Nicholson,Ganesh Raghu,Benoit Wallaert,Christian Brambilla,Kevin K. Brown,Andrew L. Cherniaev,Ulrich Costabel,David B. Coultas,Gerald S. Davis,Maurits G. Demedts,William W. Douglas,Jim J. Egan,Anders Eklund,Leonarda M. Fabbri,Craig A. Henke,Richard Hubbard,Y. Inoue,Takateru Izumi,H. M. Jansen,Ian Johnston,Dong Soon Kim,Nasreen Khalil,Fiona R. Lake,Giuseppe Lungarella,Joseph P. Lynch,Douglas W. Mapel,Fernando J. Martinez,Richard A. Matthay,Lee S. Newman,Paul W. Noble,Ken Ohta,Dario Olivieri,Luis A. Ortiz,Venerino Poletti,Robert Rodriguez-Roisin,William N. Rom,Jay Hoon Ryu,Paulo Hilário Nascimento Saldiva,Raúl H Sansores,Marvin L. Schwarz,Moisés Selman,Cecelia M. Smith,Zhaohui Tong,Zarir F Udwadia,Dominique Valeyre,Athol U. Wells,Robert A. Wise,Antonio Xaubet,Emilio Alvarez Fernandez,Elisabeth Brambilla,Vera Luiza Capelozzi,Andrew Cherniaev,Peter Dalquen,Gerhard Dekan,Philip S. Hasleton,James C. Hogg,N. A. Jambhekar,Anna Luise A Katzenstein,Michael Koss,Osamu Matsubara,Klaus Michael Müller,F. B.J.M. Thunnissen,James A. Waldron,Wei Hua Li,Paul J. Friedman,Martin Remy-Jardin,Theresa C. McLoud +82 more
TL;DR: The Diagnostic Process Is Dynamic Clinical Evaluation Radiological Evaluation Role of Surgical Lung Biopsy Unclassifiable Interstitial Pneumonia Bronchoalveolar Lavage Fluid Evaluation Idiopathic Pulmonary Fibrosis.
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Activation of the ATF6, XBP1 and grp78 genes in human hepatocellular carcinoma: a possible involvement of the ER stress pathway in hepatocarcinogenesis
Masahiro Shuda,Nobuo Kondoh,Nobuo Imazeki,Kenji Tanaka,Tetsuya Okada,Kazutoshi Mori,Akiyuki Hada,Masaaki Arai,Toru Wakatsuki,Osamu Matsubara,Naoki Yamamoto,Mikio Yamamoto +11 more
TL;DR: The endoplasmic reticulum stress pathway mediated by ATF6 and by IRE1-XBP1 systems seems essential for the transformation-associated expression of the grp78 gene in HCCs.
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The correlation between cytoplasmic overexpression of epidermal growth factor receptor and tumor aggressiveness: poor prognosis in patients with pancreatic ductal adenocarcinoma.
Shigeto Ueda,Sho Ogata,Hitoshi Tsuda,Nobuaki Kawarabayashi,Mikihiko Kimura,Yoshiaki Sugiura,Seiichi Tamai,Osamu Matsubara,Kazuo Hatsuse,Hidetaka Mochizuki +9 more
TL;DR: It is suggested that the cytoplasmic overexpression of EGFr plays a significant role in the progression of pancreatic ductal adenocarcinoma, especially in the invasion and acquisition of aggressive clinical behavior.
Journal Article
Identification and Characterization of Genes Associated with Human Hepatocellular Carcinogenesis
Nobuo Kondoh,Toru Wakatsuki,Akihide Ryo,Akiyuki Hada,Tsukasa Aihara,Sankichi Horiuchi,Narihide Goseki,Osamu Matsubara,Kenji Takenaka,Mizue Shichita,Kenji Tanaka,Masahiro Shuda,Mikio Yamamoto +12 more
TL;DR: Eight cDNAs encoding galectin 4 (Gal-4), UGT2B4 (UDP-glucuronosyltransferase), ribosomal phosphoprotein P0 (rpP0), dek, insulin-like growth factor binding protein (IGFBP) 1, vitronectin, retinoic acid-induced gene E (RIG-E), and CYP3A4 (cytochrome P450 nifedipine oxid
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Loss of ARID1A protein expression occurs as an early event in ovarian clear-cell carcinoma development and frequently coexists with PIK3CA mutations.
TL;DR: The results suggest that loss of ARID1A protein expression occurs as a very early event in ovarian clear-cell carcinoma development, similar to the pattern of PIK3CA mutation recently reported by the group, and frequently coexists (not mutually exclusive) with Pik3CA mutations.