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Patrick Hivin

Researcher at Centre national de la recherche scientifique

Publications -  8
Citations -  713

Patrick Hivin is an academic researcher from Centre national de la recherche scientifique. The author has contributed to research in topics: Transcription (biology) & Transcription factor. The author has an hindex of 8, co-authored 8 publications receiving 671 citations. Previous affiliations of Patrick Hivin include University of Montpellier.

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Human T-Cell Leukemia Virus Type 1 (HTLV-1) bZIP Protein Interacts with the Cellular Transcription Factor CREB To Inhibit HTLV-1 Transcription

TL;DR: HBZ reduces HTLV-1 transcription and virion production, and the interaction between HBZ and the cellular transcription factor CREB is characterized, suggesting that HBZ may deregulate CREB-dependent cellular gene expression.
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HTLV-I antisense transcripts initiating in the 3'LTR are alternatively spliced and polyadenylated

TL;DR: It is shown that HBZ-encoding transcripts initiate in the 3' long terminal repeat (LTR) at several positions and consist of two alternatively spliced variants (SP1 and SP2), which conclusively demonstrate the existence of antisense transcription in retroviruses.
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HBZ interacts with JunD and stimulates its transcriptional activity.

TL;DR: It is shown that HBZ can activate JunD‐dependent transcription and that its amino‐terminus is required, and that this association occurs via the bZIP domain of the two proteins.
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Nuclear localization of HTLV-I bZIP factor (HBZ) is mediated by three distinct motifs

TL;DR: It is reported here that the nuclear targeting of HBZ is mediated by three distinct nuclear localization signals and that at least two are necessary for the translocation ofHBZ to the nucleus.
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The HBZ-SP1 isoform of human T-cell leukemia virus type I represses JunB activity by sequestration into nuclear bodies

TL;DR: HBZ-mediated sequestration of JunB to the HBZ-NBs may be causing the repression of Jun B activity in vivo, and it is suggested that HBZ needs cellular partners, including bZIP factors, to form HBZ -NBs.