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Paul C. Grimm

Researcher at Stanford University

Publications -  104
Citations -  4986

Paul C. Grimm is an academic researcher from Stanford University. The author has contributed to research in topics: Transplantation & Kidney transplantation. The author has an hindex of 31, co-authored 97 publications receiving 4625 citations. Previous affiliations of Paul C. Grimm include University of Manitoba & University of California, Los Angeles.

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Banff '05 Meeting Report: differential diagnosis of chronic allograft injury and elimination of chronic allograft nephropathy ('CAN').

TL;DR: The 8th Banff Conference on Allograft Pathology was held in Edmonton, Canada, 15–21 July 2005, and major outcomes included the elimination of the non‐specific term ‘chronic allograft nephropathy’ (CAN) and the recognition of the entity of chronic antibody‐mediated rejection.
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Beneficial effects of treatment of early subclinical rejection: a randomized study.

TL;DR: It is suggested that early protocol biopsies and the treatment of subclinical rejection with corticosteroids may lead to better histologic and functional outcomes in renal transplant recipients.
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Neointimal and tubulointerstitial infiltration by recipient mesenchymal cells in chronic renal-allograft rejection

TL;DR: The presence of mesenchymal cells of host origin in the vascular and interstitial compartments of renal allografts undergoing chronic rejection provides evidence that a circulating mesenchyal precursor cell has the potential to migrate to areas of inflammation.
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Morphometric and Visual Evaluation of Fibrosis in Renal Biopsies

TL;DR: Considering efficiency, reproducibility, and functional correlation, two current techniques stand out as potentially the best for clinical trials: collagen III morphometry and visual assessment of trichrome-stained slides.
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Cell senescence in rat kidneys in vivo increases with growth and age despite lack of telomere shortening.

TL;DR: It is concluded that cell senescence occurs in both growth and aging in rat kidney and may contribute to the age-related pathology, which may reflect cumulative environmental stress.