Paul Picton

TL;DR: This negative trial could not detect a difference in the incidence of definite awareness or recovery variables between monitoring protocols based on either BIS values or anesthetic concentration.

TL;DR: Ketamine anesthesia correlates most clearly with distinct changes in the theta bandwidth, including increased power and functional connectivity, suggesting a dose-dependent effect.

TL;DR: It is demonstrated that the partial phase locking at criticality shapes the functional connectivity and asymmetric anterior‐posterior PLE topography, with low (high) PLE for high (low) degree nodes, which differentiates various pharmacologic and pathologic states of consciousness.

TL;DR: Ketamine induces altered states of consciousness during periods of reduced alpha power in the precuneus and temporal‐parietal junction, which are candidate mechanisms of the psychoactive effects of ketamine.

TL;DR: Network efficiency was significantly depressed and network clustering coefficient was significantly increased during unconsciousness; these graph theoretical measures returned to baseline during the 3 h recovery period and Posterior alpha power may be a potential biomarker for normal recovery of functional brain networks after general anesthesia.