P
Pontus B. Persson
Researcher at Humboldt University of Berlin
Publications - 266
Citations - 6933
Pontus B. Persson is an academic researcher from Humboldt University of Berlin. The author has contributed to research in topics: Kidney & Angiotensin II. The author has an hindex of 42, co-authored 244 publications receiving 6074 citations. Previous affiliations of Pontus B. Persson include Free University of Berlin & Uppsala University.
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Journal ArticleDOI
Pathophysiology of contrast medium-induced nephropathy.
TL;DR: Many experimental studies provide evidence for a greater perturbation in renal functions by dimeric contrast media in comparison to nonionic monomers as the widely used nonionic low osmolar contrast media.
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The neurovascular unit - concept review.
TL;DR: The concepts of NVU, the coupling mechanisms and research strategies are reviewed, which are one of the fundamental mechanisms for the central nervous system homeostasis.
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Contrast-induced kidney injury: Mechanisms, risk factors, and prevention
TL;DR: A unifying theory as to how CIAKI comes about is put forward; the kidney medulla's unique hyperosmolar environment concentrates CM in the tubules and vasculature, which reduces fluid viscosity and reduces cytotoxic effects.
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Comparison of various techniques used to estimate spontaneous baroreflex sensitivity (the EuroBaVar study)
Dominique Laude,Jean Luc Elghozi,Arlette Girard,Elisabeth Bellard,Malika Bouhaddi,Paolo Castiglioni,Catherine Cerutti,Andrei Cividjian,Marco Di Rienzo,Jacques Olivier Fortrat,Ben J. A. Janssen,John M. Karemaker,Georges Lefthériotis,Gianfranco Parati,Pontus B. Persson,Alberto Porta,Luc Quintin,Jacques Regnard,Heinz Rüdiger,Harald M. Stauss +19 more
TL;DR: The discrepancies between procedures show that the choice of parameters and data handling should be considered before BRS estimation, and new techniques with this set of results are needed.
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Understanding and preventing contrast-induced acute kidney injury
TL;DR: This work highlights common triggers that prompt the development of CIAKI and the subsequent mechanisms that ultimately cause kidney damage and discusses effective protective measures, such as rapidly acting oral hydration schemes and loop diuretics, in the context ofCIAKI pathophysiology.