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Richard B. Scott

Researcher at John Radcliffe Hospital

Publications -  33
Citations -  2427

Richard B. Scott is an academic researcher from John Radcliffe Hospital. The author has contributed to research in topics: Deep brain stimulation & Neuropsychology. The author has an hindex of 22, co-authored 33 publications receiving 2274 citations. Previous affiliations of Richard B. Scott include University of Oxford.

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Deep brain stimulation plus best medical therapy versus best medical therapy alone for advanced Parkinson's disease (PD SURG trial): a randomised, open-label trial

TL;DR: At 1 year, surgery and best medical therapy improved patient self-reported quality of life more than best medical Therapy alone in patients with advanced Parkinson's disease, but surgery is not without risk and targeting of patients most likely to benefit might be warranted.
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Neuropsychological, neurological and functional outcome following pallidotomy for Parkinson's disease. A consecutive series of eight simultaneous bilateral and twelve unilateral procedures.

TL;DR: Patients' perceptions of reduced postoperative functional disability and improvements in 'quality of life' also achieved statistical significance on a number of both physical and psychosocial questionnaire subscales.
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Lateralized cognitive deficits in children following cerebellar lesions.

TL;DR: It is suggested that lateralized cerebellar damage may selectively impair the development of cognitive functions subserved by the contralateral cerebral hemisphere and, in addition, that all children with Cerebellar lesions in early childhood should routinely undergo long-term monitoring of their intellectual development.
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Potentially adaptive functional changes in cognitive processing for patients with multiple sclerosis and their acute modulation by rivastigmine.

TL;DR: It is suggested that recruitment of medial prefrontal cortex is a form of adaptive brain plasticity that compensates, in part, for relative deficits in processing related to the reduced right prefrontal cortex activity with multiple sclerosis.
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Involvement of the medial pallidum in focal myoclonic dystonia: A clinical and neurophysiological case study

TL;DR: The phasic pallidal activity correlated with and led the myoclonic muscle activity, and the myoclonus was suppressed by bilateral pallidal DBS, suggesting that the medial pallidum was involved in the generation of theMyoclonic activity.