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Richard D. Palmiter
Researcher at University of Washington
Publications - 455
Citations - 74646
Richard D. Palmiter is an academic researcher from University of Washington. The author has contributed to research in topics: Dopamine & Gene. The author has an hindex of 141, co-authored 444 publications receiving 69977 citations. Previous affiliations of Richard D. Palmiter include University of Pennsylvania & Howard Hughes Medical Institute.
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Antisense oligodeoxynucleoside methylphosphonate inhibition of mouse c-myc p65 protein expression in E mu-c-myc transgenic mice.
Erica L. Wickstrom,Thomas A. Bacon,Wickstrom El,Werking Cm,Richard D. Palmiter,Ralph L. Brinster,Eric P. Sandgren +6 more
TL;DR: In transgenic mice bearing a murine immunoglobulin enhancer/c- myc fusion transgene (E mu-myc), it was found that methylphosphonates do not induce acute toxicity following intravenous administration of a 300 nmol dose.
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Neonatal 6-hydroxydopamine administration to mice is fatal.
Michelle D. Brot,Mark S. Szczypka,Randy Reavell,Brett T. Marck,Alvin M. Matsumoto,Richard D. Palmiter +5 more
TL;DR: The hypothesis that killing dopaminergic neurons with 6-OHDA might promote survival of dopamine-deficient mice was tested and it was demonstrated that regardless of genotype or environmental temperature, bilateral neonatal 6- OHDA lesions are lethal to mice.
Posted ContentDOI
NKB Signaling in the Medial Amygdala Stimulates Gonadotropin Release in a Kisspeptin-Independent Manner in Female Mice
Chrysanthi Fergani,Chrysanthi Fergani,Silvia Leon,Silvia Leon,Stephanie L Padilla,Anne M.J. Verstegen,Anne M.J. Verstegen,Richard D. Palmiter,Víctor M. Navarro,Víctor M. Navarro +9 more
TL;DR: It is demonstrated that in the presence of E2, NKB signaling induces LH release in a kisspeptin-independent manner, and senktide (NKB receptor agonist) delivery to the medial amygdala (MeA) increases LH in E2-treated Kiss1 KO females similar to controls, and thus, this increase is independent of Kiss1 neurons.
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Fast-acting neurons that suppress appetite.
TL;DR: Recent studies reveal several groups of neurons that become activated upon anticipation or consumption of meals, which constitute key components of the complex feedback system that prevents continuous feeding by mice.
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Topographic representation of current and future threats in the mouse nociceptive amygdala
Anna J Bowen,Y Waterlily Huang,Jane Y. Chen,Jordan L. Pauli,Carlos A. Campos,Richard D. Palmiter +5 more
TL;DR: In this article , the authors found that a primary nociceptive population within the central amygdala (CeA) of mice, defined by CGRP-receptor (Calcrl) expression, receives topographic sensory information, with spatially defined representations of internal and external stimuli.