R
Richard I. Enelow
Researcher at Dartmouth College
Publications - 31
Citations - 2451
Richard I. Enelow is an academic researcher from Dartmouth College. The author has contributed to research in topics: Cytotoxic T cell & CD8. The author has an hindex of 24, co-authored 31 publications receiving 2127 citations. Previous affiliations of Richard I. Enelow include Northwestern University & Dartmouth–Hitchcock Medical Center.
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Journal ArticleDOI
Critical Role of IL-17RA in Immunopathology of Influenza Infection
Christopher R. Crowe,Kong Chen,Derek Pociask,John F. Alcorn,Cameron Krivich,Richard I. Enelow,Ted M. Ross,Joseph L. Witztum,Jay K. Kolls +8 more
TL;DR: Data support targeting IL-17 orIL-17RA in acute lung injury due to acute viral infection and shows significantly reduced levels of oxidized phospholipids, which have previously been shown to be an important mediator in several models of acute Lung injury, including influenza infection and gastric acid aspiration.
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Influenza A Inhibits Th17-Mediated Host Defense against Bacterial Pneumonia in Mice
Anupa Kudva,Erich V. Scheller,Keven M. Robinson,Chris R. Crowe,Sun Mi Choi,Samantha Slight,Shabaana A. Khader,Patricia J. Dubin,Richard I. Enelow,Jay K. Kolls,Jay K. Kolls,John F. Alcorn +11 more
TL;DR: Data indicate a novel mechanism by which influenza A-induced type I IFNs inhibit Th17 immunity and increase susceptibility to secondary bacterial pneumonia.
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Low-Dose Arsenic Compromises the Immune Response to Influenza A Infection in Vivo
TL;DR: The data indicate that chronic As exposure significantly compromises the immune response to infection, and Alterations in response to repeated lung infection may also contribute to other chronic illnesses, such as bronchiectasis, which is elevated by As exposure in epidemiology studies.
Journal ArticleDOI
IL-22 Is Essential for Lung Epithelial Repair following Influenza Infection
Derek Pociask,Erich V. Scheller,Sivanarayana Mandalapu,Kevin J. McHugh,Richard I. Enelow,Cheryl L. Fattman,Jay K. Kolls,John F. Alcorn +7 more
TL;DR: Gene expression analysis revealed aberrant expression of epithelial genes involved in repair processes, among changes in several other biological processes, which indicate that IL-22 is required for normal lung repair after influenza infection.
Journal ArticleDOI
In utero arsenic exposure and infant infection in a United States cohort: A prospective study ☆
Shohreh F. Farzan,Susan A. Korrick,Susan A. Korrick,Zhigang Li,Richard I. Enelow,A. Jay Gandolfi,Juliette C. Madan,Juliette C. Madan,Kari C. Nadeau,Margaret R. Karagas +9 more
TL;DR: Initial evidence is provided that in utero As exposure may be related to infant infection and infection severity and provide insight into the early life impacts of fetal As exposure.