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Rosa Maria Borzì

Researcher at I.O.R.

Publications -  71
Citations -  3757

Rosa Maria Borzì is an academic researcher from I.O.R.. The author has contributed to research in topics: Chondrocyte & Cellular differentiation. The author has an hindex of 28, co-authored 69 publications receiving 3164 citations. Previous affiliations of Rosa Maria Borzì include University of Bologna.

Papers
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Journal ArticleDOI

NF-κB Signaling: Multiple Angles to Target OA

Kenneth B. Marcu, +4 more
- 01 May 2010 - 
TL;DR: Growing evidence points to NF-kappaB signaling as not only playing a central role in the pro-inflammatory stress-related responses of chondrocytes to extra- and intra-cellular insults, but also in the control of their differentiation program.
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Roles of inflammatory and anabolic cytokines in cartilage metabolism: signals and multiple effectors converge upon MMP-13 regulation in osteoarthritis

Mary B. Goldring, +10 more
- 24 Feb 2011 - 
TL;DR: A thorough understanding of the similarities and particularly the marked differences in mechanisms of cartilage remodeling during development, osteoarthritis, and aging may lead to more effective strategies for preventing cartilage damage and promoting repair.
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Biomaterials: Foreign Bodies or Tuners for the Immune Response?

Erminia Mariani, +3 more
- 01 Feb 2019 - 
TL;DR: Recent research breakthroughs have provided a broader insight on the correct choice of biomaterial physicochemical modifications to tune the reaction of the host immune system to implanted biomaterial and to favor integration and healing.
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Enhanced and coordinated in vivo expression of inflammatory cytokines and nitric oxide synthase by chondrocytes from patients with osteoarthritis

Cinzia Melchiorri, +8 more
- 01 Dec 1998 - 
TL;DR: The enhanced and coordinated expression of IL-1beta, TNFalpha, and iNOS by chondrocytes strongly supports the hypothesis that chondROcytes are the major site of production of mediators of inflammation in human OA, thus playing a primary role in the pathogenesis of this disease.
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p16INK4a and its regulator miR-24 link senescence and chondrocyte terminal differentiation-associated matrix remodeling in osteoarthritis

Didier Philipot, +20 more
- 27 Feb 2014 - 
TL;DR: In this article, the role of the senescence marker p16INK4a and its regulation by microRNAs (miRs) during OA and terminal chondrogenesis was evaluated.