R
Rosanna Mazzolla
Researcher at University of Perugia
Publications - 41
Citations - 2273
Rosanna Mazzolla is an academic researcher from University of Perugia. The author has contributed to research in topics: Candida albicans & Cryptococcus neoformans. The author has an hindex of 20, co-authored 41 publications receiving 2152 citations. Previous affiliations of Rosanna Mazzolla include University of Modena and Reggio Emilia.
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Immortalization of murine microglial cells by a v-raf / v-myc carrying retrovirus
TL;DR: Since BV-2 cells retain most of the morphological, phenotypical and functional properties described for freshly isolated microglial cells, it can be concluded that J2 virus infection has resulted in the immortalization of active microglia cells.
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An immortalized cell line expresses properties of activated microglial cells.
TL;DR: The immortalized cell line (BV‐2) shares properties with body macrophages with respect to the antigen profile, their phagocytic capacity and antimicrobial activity, and represents a suitable model for in vitro studies of activated microglial cells.
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A multicentre evaluation of the accuracy and performance of IP-10 for the diagnosis of infection with M. tuberculosis.
Morten Ruhwald,José Domínguez,Irene Latorre,Monica Losi,Luca Richeldi,Maria Bruna Pasticci,Rosanna Mazzolla,Delia Goletti,Ornella Butera,Judith Bruchfeld,Hans Gaines,Irini Gerogianni,Tamara Tuuminen,Giovanni Ferrara,Jesper Eugen-Olsen,Pernille Ravn +15 more
TL;DR: Previous findings are confirmed and validates and substance is added to IP-10 as a novel diagnostic marker for infection with M. tuberculosis; further studies are needed to test the clinical impact of these findings.
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Role of nitric oxide and melanogenesis in the accomplishment of anticryptococcal activity by the BV-2 microglial cell line
Elisabetta Blasi,R. Barluzzi,Rosanna Mazzolla,Brunella Tancini,Silvia Saleppico,Manuela Puliti,Lucia Pitzurra,Francesco Bistoni +7 more
TL;DR: Evidence is provided that NO-dependent events are involved in the fulfillment of anticryptococcal activity by activated microglial cells and that fungal melanization is a precious escamotage through which C. neoformans overcomes host defenses.
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Experimental model of intracerebral infection with Cryptococcus neoformans: roles of phagocytes and opsonization.
TL;DR: In vitro data showed that microglial cells were proficient anticryptococcal effectors, provided opsonized microorganisms were used, and phagocyte-mediated mechanisms are likely involved in local resistance to i.c.C. infection with C. neoformans.