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Rosemarie W. Hammond

Researcher at United States Department of Agriculture

Publications -  111
Citations -  2700

Rosemarie W. Hammond is an academic researcher from United States Department of Agriculture. The author has contributed to research in topics: Viroid & Potato spindle tuber viroid. The author has an hindex of 27, co-authored 106 publications receiving 2378 citations. Previous affiliations of Rosemarie W. Hammond include Agricultural Research Service & Johns Hopkins University.

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The Apache Point Observatory Galactic Evolution Experiment (APOGEE) Spectrographs

John C. Wilson, +91 more
TL;DR: The Apache Point Observatory Galactic Evolution Experiment (APOGEE) as mentioned in this paper is a survey of ~ 10^5 red giant stars that systematically sampled all Milky Way populations (bulge, disk, and halo) to study the Galaxy's chemical and kinematical history.
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Cell-to-cell movement of potato spindle tuber viroid

TL;DR: Data indicate that PSTVd moves from cell to cell via plasmodesmata, and this movement may be mediated by a specific sequence or structural motif.
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Identification of multiple structural domains regulating viroid pathogenicity.

TL;DR: Quantitative comparisons of symptom development and progeny accumulation revealed that the TL domain of TASVd contains a determinant required for appearance of severe veinal necrosis in tomato, and the severe epinasty and stunting characteristic of TAsVd requires the presence of its TL and P domains.
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Molecular biology of viroid-host interactions and disease control strategies.

TL;DR: Efforts to engineer viroid resistance into host species have been underway for several years, and include the use of antisenseRNA, antisense RNA plus ribozymes, a dsRNase, and siRNAs, among others.
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RNA structural features responsible for potato spindle tuber viroid pathogenicity.

TL;DR: Comparison of the biological and structural properties of 12 representative PSTVd sequence variants point to major differences in the geometry of their pathogenicity domains, which suggests alterations in RNA structure together with concomitant alterations inRNA-protein interaction(s) may be the primary cause of viroid pathogenicicity.