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S. Molin

Researcher at Technical University of Denmark

Publications -  18
Citations -  1410

S. Molin is an academic researcher from Technical University of Denmark. The author has contributed to research in topics: Biofilm & Staphylococcus epidermidis. The author has an hindex of 11, co-authored 18 publications receiving 1299 citations.

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Role of autolysin-mediated DNA release in biofilm formation of Staphylococcus epidermidis

TL;DR: Evidence is presented that extracellular DNA is important for the initial phase of biofilm development by S. epidermidis on polystyrene or glass surfaces under static or hydrodynamic conditions, and that the ext racellular DNA promotes biofilm formation of the remaining population.
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Is there a role for quorum sensing signals in bacterial biofilms

TL;DR: This work has suggested that differentiated biofilms may also be the net result of many independent interactions, rather than being determined by a particular global quorum sensing system.
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Formation and properties of in vitro biofilms of ica-negative Staphylococcus epidermidis clinical isolates

TL;DR: It is suggested that the biofilm-positive/ica-negative strain represents a newly emergent subpopulation of S. epidermidis clinical strains, arising from selection by antibiotics in the nosocomial milieu, which displays a survival advantage in its host environment.
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Pseudomonas aeruginosa extracellular products inhibit staphylococcal growth, and disrupt established biofilms produced by Staphylococcus epidermidis.

TL;DR: Findings reveal that P. aeruginosa extracellular products are important microbial competition factors that overcome competition with S. epidermidis, and the results may provide clues for the development of a novel strategy for controlling S.EpidermidIS growth in planktonic cultures.
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Coexistence and Within-Host Evolution of Diversified Lineages of Hypermutable Pseudomonas aeruginosa in Long-term Cystic Fibrosis Infections

TL;DR: Comparison of the genomes of 13 and 14 isolates of P. aeruginosa mutator populations from an Argentinian and a Danish CF patient showed extensive within-patient genomic diversification, suggesting that the genes were targeted by mutation to optimize pathogenic fitness.