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Samuel K. Ludwin
Researcher at Montreal Neurological Institute and Hospital
Publications - 69
Citations - 26812
Samuel K. Ludwin is an academic researcher from Montreal Neurological Institute and Hospital. The author has contributed to research in topics: Remyelination & Myelin. The author has an hindex of 34, co-authored 67 publications receiving 23050 citations. Previous affiliations of Samuel K. Ludwin include McGill University & Queen's University.
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Evolving concepts and issues in remyelination
TL;DR: The ability of the central nervous system to undergo remyelination is now well established in both clinical and experimental situations and the focus of investigation has shifted from simple morphologic documentation to a detailed examination of the basic biologic processes controlling or limiting remyElination.
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Glial Cells as Regulators of Neuroimmune Interactions in the Central Nervous System
TL;DR: The evolution of this field is described, from the initial work characterizing the properties of the microglia and astrocytes in isolation to the more complex challenge of defining the molecular pathways they use to interact with other cell types as well as with each other.
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Hereditary diffuse leukoencephalopathy with neuroaxonal spheroids: novel imaging findings
TL;DR: This is the first report of an abnormal apparent diffusion coefficient (ADC) map in HDLS, and a previously healthy 46-year-old man presented following a 1 year history of slurred speech and word finding difficulty that progressed until …
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The release of endogenous acetylcholine from the medial septum/diagonal band of rat brain.
TL;DR: Results indicate that ACh is released in the ms/vdB by a Ca2+-dependent and atropine-sensitive process.
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Astrocytes in the Pathogenesis of Multiple Sclerosis: An In Situ MicroRNA Study.
Vijayaraghava T.S. Rao,Shih-Chieh Fuh,Jason Karamchandani,John Woulfe,David G. Munoz,Benjamin Ellezam,Manon Blain,Ming-Kai Ho,Barry J. Bedell,Jack P. Antel,Samuel K. Ludwin +10 more
TL;DR: The regional and lesion-stage differences of expression of these miRNAs indicate the remarkable ability of astrocytes to show a wide range of selective responses in the face of differing insults and phases of resolution.