S
Sandra J. Peters
Researcher at Brock University
Publications - 55
Citations - 1470
Sandra J. Peters is an academic researcher from Brock University. The author has contributed to research in topics: Skeletal muscle & Pyruvate dehydrogenase complex. The author has an hindex of 22, co-authored 54 publications receiving 1351 citations.
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Journal ArticleDOI
Variability of triacylglycerol content in human skeletal muscle biopsy samples
TL;DR: It is concluded that the TGm store measured in repeated biopsies of human skeletal muscle is variable, with a CV of 20-26%.
Journal ArticleDOI
Prior heavy exercise elevates pyruvate dehydrogenase activity and speeds O2 uptake kinetics during subsequent moderate‐intensity exercise in healthy young adults
Brendon J. Gurd,Sandra J. Peters,George J. F. Heigenhauser,Paul J. LeBlanc,Timothy J. Doherty,Donald H. Paterson,John M. Kowalchuk +6 more
TL;DR: Findings are consistent with metabolic inertia, via delayed activation of PDH, in part limiting the adaptation of pulmonary and muscle O2 consumption during the normal transition to exercise, and suggest that greater muscle perfusion may also have contributed to the observed faster kinetics in Mod2.
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Skeletal muscle PLIN proteins, ATGL and CGI-58, interactions at rest and following stimulated contraction
TL;DR: In skeletal muscle, during contraction-induced muscle lipolysis, ATGL and CGI-58 strongly associate and that the PLIN proteins work together to regulate lipolytic activity, in part, by preventing ATGL-58 interactions at rest.
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Effects of acidosis on rat muscle metabolism and performance during heavy exercise
TL;DR: Acidosis decreased the muscle's ability to generate isometric tension and depressed both aerobic and anaerobic metabolism and Muscle creatine phosphate utilization and ATP levels were unaffected by acidosis.
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Enzymatic regulation of glucose disposal in human skeletal muscle after a high-fat, low-carbohydrate diet
TL;DR: The data demonstrate that the decreased glucose disposal during the OGTT after the 56-h HF/LC diet was in part related to decreased oxidative carbohydrate disposal in skeletal muscle and not to decreased glycogen storage.