S
Shaogen Wu
Researcher at Rutgers University
Publications - 43
Citations - 1557
Shaogen Wu is an academic researcher from Rutgers University. The author has contributed to research in topics: Dorsal root ganglion & Neuropathic pain. The author has an hindex of 19, co-authored 38 publications receiving 1067 citations. Previous affiliations of Shaogen Wu include Rowan University & Nanjing University.
Papers
More filters
Journal ArticleDOI
DNA methyltransferase DNMT3a contributes to neuropathic pain by repressing Kcna2 in primary afferent neurons
Jian-Yuan Zhao,Lingli Liang,Xiyao Gu,Zhisong Li,Zhisong Li,Shaogen Wu,Linlin Sun,Fidelis E. Atianjoh,Fidelis E. Atianjoh,Jian Feng,Kai Mo,Shushan Jia,Brianna Marie Lutz,Alex Bekker,Eric J. Nestler,Yuan Xiang Tao,Yuan Xiang Tao +16 more
TL;DR: It is reported that peripheral nerve injury increases expression of the DNA methyltransferase DNMT3a in the injured DRG neurons via the activation of the transcription factor octamer transcription factor 1.
Journal ArticleDOI
MicroRNA-181a Suppresses Mouse Granulosa Cell Proliferation by Targeting Activin Receptor IIA
TL;DR: An interplay between miR-181a and acvr2a is identified, and an important role of miR -181a in regulating granulosa cell proliferation and ovarian follicle development is revealed.
Journal ArticleDOI
MicroRNA-133b stimulates ovarian estradiol synthesis by targeting Foxl2
Anyi Dai,Haixiang Sun,Ting Fang,Qun Zhang,Shaogen Wu,Yue Jiang,Lijun Ding,Guijun Yan,Yali Hu +8 more
TL;DR: It is demonstrated that miR‐133b down‐regulates Foxl2 expression in granulosa cells by directly targeting the 3′UTR, thus inhibiting the Foxl 2‐mediated transcriptional repression of StAR and CYP19A1 to promote estradiol production.
Journal ArticleDOI
MicroRNA-145 suppresses mouse granulosa cell proliferation by targeting activin receptor IB.
TL;DR: It is shown that miR‐145 suppresses mGC proliferation by targeting ACVRIB and also interfered with activin‐induced Smad2 phosphorylation.
Journal ArticleDOI
Long noncoding RNA (lncRNA): a target in neuropathic pain.
TL;DR: Evidence of how peripheral nerve injury causes the dysregulation of lncRNAs in these pain-related regions is provided and the potential mechanisms of how dysregulated lnc RNAs contribute to the pathogenesis of neuropathic pain are discussed.