S
Shin’Ichiro Satake
Researcher at Graduate University for Advanced Studies
Publications - 13
Citations - 561
Shin’Ichiro Satake is an academic researcher from Graduate University for Advanced Studies. The author has contributed to research in topics: Inhibitory postsynaptic potential & Cerebellar cortex. The author has an hindex of 10, co-authored 13 publications receiving 513 citations. Previous affiliations of Shin’Ichiro Satake include Mitsubishi & National Institutes of Natural Sciences, Japan.
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Journal ArticleDOI
Synaptic activation of AMPA receptors inhibits GABA release from cerebellar interneurons.
TL;DR: Dual AMPA receptor-mediated functions of excitation and disinhibition may ensure transmission of cerebellar CF signals controlling sensorimotor coordination.
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Identification and Characterization of GABAA Receptor Autoantibodies in Autoimmune Encephalitis
Toshika Ohkawa,Shin’Ichiro Satake,Norihiko Yokoi,Yu Miyazaki,Tomohiko Ohshita,Gen Sobue,Hiroshi Takashima,Osamu Watanabe,Yuko Fukata,Masaki Fukata +9 more
TL;DR: Application of the patients' serum at the time of symptom presentation of encephalitis to rat hippocampal neuron cultures specifically decreased both synaptic and surface GABAA receptors, and treatment of neurons with the patients’ serum selectively reduced miniature IPSC amplitude and frequency without affecting miniature EPSCs.
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GABAB receptor-mediated presynaptic inhibition of glutamatergic and GABAergic transmission in the basolateral amygdala
TL;DR: Evidence is provided that synaptic activation of GABA(B) heteroreceptors elicits presynaptic inhibition of glutamatergic excitatory transmission in the basolateral amygdala (BLA) and the paired-pulse ratio of EPSCs was increased during the stimulation-induced inhibition.
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Enhanced inhibitory neurotransmission in the cerebellar cortex of Atp1a3-deficient heterozygous mice
Keiko Ikeda,Shin’Ichiro Satake,Tatsushi Onaka,Hiroki Sugimoto,Naoki Takeda,Keiji Imoto,Kiyoshi Kawakami +6 more
TL;DR: The findings suggest that ATP1A3 haploinsufficiency in the cerebellum has some effect on the inhibitory, but not the excitatory, circuitry and the interaction among different cell types during development.
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β-Adrenergic Receptor-Mediated Presynaptic Facilitation of Inhibitory GABAergic Transmission at Cerebellar Interneuron-Purkinje Cell Synapses
TL;DR: The data suggest that the noradrenergic facilitation of GABAergic transmission in the rat cerebellar cortex is mediated, at least in part, by depolarization and action potential discharges in the BCs through activation of the beta(2)-adrenoceptors in BCs coupled to intracellular cyclic AMP formation.