Sofie Hexeberg

TL;DR: The results indicate that docosahexaenoic acid, in contrast to eicosapentaenoic Acid, does not inhibit the synthesis and secretion of triglycerides in the liver and emphasize the importance that stimulation of peroxisomal beta-oxidation by these n-3 fatty acids is not sufficient to decrease the serum levels of triglyceride.

TL;DR: The results suggest that the instant hypolipidemia in rats given EPA could be explained at least in part by a sudden increase in mitochondrial fatty acid oxidation, thereby reducing the availability of fatty acids for lipid synthesis in the liver for export, e.g., in the form of very low density lipoproteins, even before EPA induced peroxisomal fatty Acid oxidation, reduced triglyceride biosynthesis and diminished lipogenesis.

TL;DR: EPA and DHA have different effects on mitochondrial biogenesis, as indicated by the mean area occupied by the mitochondria and the size distribution of mitochondrial areas in both fiber types were highly similar in DHA‐treated and control animals.

TL;DR: Lipid accumulation in the myocardial cells was shown by morphometry after 10 d of cholesterol feeding, without any visible damage to the tissue.

TL;DR: The presence of pectin in the diets of cholesterol-fed rats resulted in increased hepatic concentration of triacylglycerols and increased mitochondrial fatty acid oxidation, which may be mediated by a reduced efflux of Triacyl Glycerol from the liver.