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Sridevi Devaraj

Researcher at Baylor College of Medicine

Publications -  390
Citations -  23771

Sridevi Devaraj is an academic researcher from Baylor College of Medicine. The author has contributed to research in topics: Metabolic syndrome & Proinflammatory cytokine. The author has an hindex of 85, co-authored 365 publications receiving 21831 citations. Previous affiliations of Sridevi Devaraj include University of Madras & Boston Children's Hospital.

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Association of Apolipoprotein B and Nuclear Magnetic Resonance Spectroscopy–Derived LDL Particle Number with Outcomes in 25 Clinical Studies: Assessment by the AACC Lipoprotein and Vascular Diseases Division Working Group on Best Practices

TL;DR: The adoption of apo B and/or LDL-P as indicators of atherogenic particle numbers into CVD risk screening and treatment guidelines is supported because of its availability, scalability, standardization, and relatively low cost.
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Reduced-calorie orange juice beverage with plant sterols lowers C-reactive protein concentrations and improves the lipid profile in human volunteers.

TL;DR: In this paper, the effect of plant sterols on C-reactive protein (hs-CRP), the prototypic marker of inflammation, and LDL cholesterol was investigated using a reduced-calorie (50 calories/240 mL) orange juice beverage.
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Diabetes is a proinflammatory state: a translational perspective.

TL;DR: It appears that both Type 1 and Type 2 diabetes are proinflammatory states and that these could contribute to increased diabetic vasculopathies.
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The effects of alpha-tocopherol on critical cells in atherogenesis.

TL;DR: The anti-atherogenic effects of alpha tocopherol on crucial cells in atherogenesis have been highlighted, such as the inhibition of smooth muscle cell proliferation, preservation of endothelial function, inhibition of monocyte-endothelial adhesion, inhibited of monocytes reactive oxygen species and cytokine release, and inhibition of platelet adhesion and aggregation.
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The airway microbiome of intubated premature infants: characteristics and changes that predict the development of bronchopulmonary dysplasia.

TL;DR: The airways of premature infants are not sterile at birth and reduced diversity of the microbiome may be an important factor in the development of BPD and is not associated with differences in inflammatory mediators.