scispace - formally typeset
S

Stanko S. Stojilkovic

Researcher at National Institutes of Health

Publications -  248
Citations -  11130

Stanko S. Stojilkovic is an academic researcher from National Institutes of Health. The author has contributed to research in topics: Receptor & Gonadotropic cell. The author has an hindex of 58, co-authored 240 publications receiving 10456 citations. Previous affiliations of Stanko S. Stojilkovic include University of California, Davis & Academy of Sciences of the Czech Republic.

Papers
More filters
Journal ArticleDOI

Activation and Regulation of Purinergic P2X Receptor Channels

TL;DR: Data obtained from numerous site-directed mutagenesis experiments accumulated during the last 15 years are discussed with reference to the crystal structure, allowing a structural interpretation of the molecular basis of orthosteric and allosteric ligand actions.
Journal ArticleDOI

Gonadotropin-Releasing Hormone Receptors: Structure and Signal Transduction Pathways

TL;DR: The review addressed the interactions between cytoplasmic calcium and protein kinase C (PKC) in the control of gonadotropin synthesis and release, as well as the participation of phospholipase D (PLD), arachidonic acid (AA) and its metabolites in GnRHinduced signaling.
Journal ArticleDOI

Gigantism and Acromegaly Due to Xq26 Microduplications and GPR101 Mutation

TL;DR: A pediatric disorder is described that is caused by an Xq26.3 genomic duplication and is characterized by early-onset gigantism resulting from an excess of growth hormone.
Journal ArticleDOI

F(ab)'2-mediated neutralization of C3a and C5a anaphylatoxins: a novel effector function of immunoglobulins.

TL;DR: Molecular modeling, surface plasmon resonance (SPR) and western blot analyses suggested a physical association between anaphylatoxins and the constant region of F(ab)′2 that could interfere with the role of C3a and C5a in inflammation.
Journal ArticleDOI

Ion Channels and Signaling in the Pituitary Gland

TL;DR: This review summarizes recent findings in this field and current understanding of the complex relationship between voltage-gated ion channels, ligand- gated ion channel channels, gap junction channels, and G protein-coupled receptors in pituitary cells.