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Sunir J. Garg

Researcher at Wills Eye Institute

Publications -  207
Citations -  3978

Sunir J. Garg is an academic researcher from Wills Eye Institute. The author has contributed to research in topics: Medicine & Endophthalmitis. The author has an hindex of 32, co-authored 157 publications receiving 3017 citations. Previous affiliations of Sunir J. Garg include University of Michigan & California Pacific Medical Center.

Papers
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Journal ArticleDOI

Complications of intravitreal injections.

TL;DR: The overall risk of endophthalmitis and retinal detachment appears to be low and most of the commonly used drugs are well tolerated, even with repeat injection.
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Outcomes and risk factors associated with endophthalmitis after intravitreal injection of anti-vascular endothelial growth factor agents.

TL;DR: Most patients in whom presumed infectious endophthalmitis develop after anti-VEGF injection regained baseline vision after treatment, and a low threshold for vitreous tap with intravitreal antibiotic injection may be warranted.
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The Role of Topical Antibiotic Prophylaxis to Prevent Endophthalmitis after Intravitreal Injection

TL;DR: Using postinjection topical antibiotic drops does not reduce the risk of endophthalmitis developing and is associated with a trend toward higher incidence of endophysiology, but topical antibiotic use was associated with increased risk of suspected endophhalmitis.
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Subretinal Hyperreflective Material in the Comparison of Age-Related Macular Degeneration Treatments Trials

Alex S. Willoughby, +861 more
- 01 Sep 2015 - 
TL;DR: In eyes with neovascular age-related macular degeneration (AMD), SHRM is common and often persists after anti-vascular endothelial growth factor treatment, and greater SHRM dimensions were associated with worse VA.
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Changes in Antibiotic Resistance Patterns of Conjunctival Flora Due to Repeated Use of Topical Antibiotics after Intravitreal Injection

TL;DR: Repeated use of topical fluoroquinolones after intravitreal injections may have a detrimental effect on eye health by breeding resistance in the bacterial flora.