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Takanori Ida

Researcher at University of Miyazaki

Publications -  59
Citations -  2723

Takanori Ida is an academic researcher from University of Miyazaki. The author has contributed to research in topics: Ghrelin & Neuromedin U. The author has an hindex of 23, co-authored 53 publications receiving 2471 citations. Previous affiliations of Takanori Ida include Kurume University.

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Effect of lateral cerebroventricular injection of the appetite-stimulating neuropeptide, orexin and neuropeptide Y, on the various behavioral activities of rats.

TL;DR: The results suggest that orexin may be involved in the regulation of several other behavioral activities in rats, besides feeding.
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Possible involvement of orexin in the stress reaction in rats

TL;DR: It is suggested that CRF is involved in orexin-induced behaviors, and that orex in may play an important role in some stress reactions.
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Role for central ghrelin in food intake and secretion profile of stomach ghrelin in rats.

TL;DR: The results suggest that endogenous centrally released ghrelin participates in the regulation of food intake and body weight, acyl Ghrelin is secreted from the stomach, intestinal hormones stimulate gh Relin release from theomach, and regulation of the diurnal rhythm of ghrelIn is complex, since ghrel in secretion is augmented under conditions of both gastric emptying and filling.
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Peripheral ghrelin transmits orexigenic signals through the noradrenergic pathway from the hindbrain to the hypothalamus.

TL;DR: It is demonstrated that peripheral ghrelin signaling, which travels to the nucleus tractus solitarius (NTS) at least in part via the vagus nerve, increases noradrenaline (NA) in the arcuate nucleus of the hypothalamus, thereby stimulating feeding at least partially through alpha-1 and beta-2noradrenergic receptors.
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Central control of bone remodeling by neuromedin U.

TL;DR: Physiological and cell-based assays indicate that NMU acts in the central nervous system, rather than directly on bone cells, to regulate bone remodeling, and suggest that it may be the first central mediator of leptin-dependent regulation of bone mass identified to date.