Taro Arima

TL;DR: This review will address the current understanding of classification issues, epidemiology and neurobiological mechanisms of craniofacial pain, and enable clinicians to understand the reasons why simple cause-effect relationships between bruxism and cranioFacial pain are inadequate and the current implications for management of cranioskeletal pain.

TL;DR: It is suggested that significant but low levels of postexercise muscle soreness can be elicited by standardized grinding movements in the masticatory system of healthy subjects.

TL;DR: The results suggest that tonic saline-induced jaw-muscle pain is able to modulate the motor unit recruitment pattern of the jaw-closing muscles on the painful side and an inhibition of static EMG activity is suggested.

TL;DR: The results showed that the time spent in SWS was significantly decreased during the first sleep deprivation night, but there were no significant effects on nocturnal EMG activity, suggesting that deprivation of SWS may not interact immediately withNocturnal jaw-Muscle activity and jaw-muscle pain.

TL;DR: It is suggested that there are no major differences between patients with different craniofacial pain conditions and pain-free individuals in terms of jaw-muscle EMG activity recorded with a single-channel EMG device during sleep, however, some associations may exist between the level ofEMG activity and various parameters of cranioFacial pain.