T
Theodore A. Craig
Researcher at Mayo Clinic
Publications - 47
Citations - 1763
Theodore A. Craig is an academic researcher from Mayo Clinic. The author has contributed to research in topics: Calcitriol receptor & Sclerostin. The author has an hindex of 21, co-authored 46 publications receiving 1612 citations.
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Journal ArticleDOI
Secreted frizzled-related protein 4 is a potent tumor-derived phosphaturic agent
Theresa J. Berndt,Theodore A. Craig,Ann E. Bowe,John Vassiliadis,David Reczek,Richard Finnegan,Suzanne M. Jan de Beur,Susan C. Schiavi,Rajesh Kumar +8 more
TL;DR: SFRP-4 displays phosphatonin-like properties, because it is a circulating protein that promotes phosphaturia and hypophosphatemia and blunts compensatory increases in 1alpha, 25-dihydroxyvitamin D.
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Evidence for a signaling axis by which intestinal phosphate rapidly modulates renal phosphate reabsorption
Theresa J. Berndt,Leslie F. Thomas,Theodore A. Craig,Stacy Sommer,Xujian Li,Eric J. Bergstralh,Rajesh Kumar +6 more
TL;DR: The experiments demonstrate the presence of a previously unrecognized phosphate gut–renal axis that rapidly modulates renal phosphate excretion after the intestinal administration of phosphate.
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1α,25-Dihydroxyvitamin D3 Regulates Mitochondrial Oxygen Consumption and Dynamics in Human Skeletal Muscle Cells
Zachary C. Ryan,Theodore A. Craig,Clifford D.L. Folmes,Xuewei Wang,Ian R. Lanza,Niccole Schaible,Jeffrey L. Salisbury,K. Sreekumaran Nair,Andre Terzic,Gary C. Sieck,Rajiv Kumar +10 more
TL;DR: 1α,25(OH)2D3 regulates mitochondrial function, dynamics, and enzyme function, which is likely to influence muscle strength and vitamin D deficiency and chronic renal failure in skeletal muscle cells.
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Sclerostin alters serum vitamin D metabolite and fibroblast growth factor 23 concentrations and the urinary excretion of calcium.
Zachary C. Ryan,Hemamalini Ketha,Melissa S. McNulty,Meghan E. McGee-Lawrence,Theodore A. Craig,Joseph P. Grande,Jennifer J. Westendorf,Ravinder J. Singh,Rajiv Kumar +8 more
TL;DR: The data show that sclerostin not only alters bone mineralization, but also influences mineral metabolism by altering concentrations of hormones that regulate mineral accretion.
Journal ArticleDOI
The phosphatonins and the regulation of phosphate transport and vitamin D metabolism.
TL;DR: Interestingly, despite the induction of hypophosphatemia by FGF-23 and sFRP-4 in vivo, serum 1, 25-dihydroxyvitamin D (1alpha,25(OH)(2)D) concentrations are decreased or remain inappropriately normal, suggesting an inhibitory effect of these proteins on 25-hydroxyv Vitamin D 1alpha-hydroxylase activity.