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Ulrich Bogdahn

Researcher at University of Regensburg

Publications -  347
Citations -  36060

Ulrich Bogdahn is an academic researcher from University of Regensburg. The author has contributed to research in topics: Neural stem cell & Neurogenesis. The author has an hindex of 67, co-authored 344 publications receiving 32279 citations. Previous affiliations of Ulrich Bogdahn include Hoffmann-La Roche & Volkswagen Foundation.

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Diabetes, use of metformin, and the risk of meningioma.

TL;DR: A matched case-control analysis using data from the U.K.-based Clinical Practice Research Datalink to analyse diabetes status, duration of diabetes, glycemic control, and use of metformin, sulfonylureas, and insulin in relation to the risk of meningioma found the suggestion of an inverse association.
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In vitro studies of cytokine-mediated interactions between malignant glioma and autologous peripheral blood mononuclear cells

TL;DR: It is concluded that glioma-lymphocyte growth regulatory networks include stimulatory and inhibitory factors from both cell populations, which may modulate tumor progression and have relevance for adoptive immunotherapy in patients with gliomas.
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The impact of white matter lesions on the cognitive outcome of subthalamic nucleus deep brain stimulation in Parkinson’s disease

TL;DR: Damaged white matter may lead to a reduced compensation of disconnections in cognitive circuits caused by the implantation of the DBS electrodes or by chronic stimulation and the role of WML as a prognostic factor for the cognitive outcome after DBS may be underestimated.
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Intracranial hemorrhage: frequency, location, and risk factors identified in a TeleStroke network.

TL;DR: The relatively low proportion of interhospital transfers shown in this study reflects a differentiated decision process on the basis of both guidelines and standard operating procedures.
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Selective bilateral hippocampal lesions after theophylline-induced status epilepticus causes a permanent amnesic syndrome.

TL;DR: Evidence is provided that theophylline has the potential to provoke permanent seizure-induced neural damage, presumably via inhibition of adenosine receptors, and especially in vulnerable regions of the brain, such as the hippocampus.