V
Vojo Deretic
Researcher at University of New Mexico
Publications - 278
Citations - 51981
Vojo Deretic is an academic researcher from University of New Mexico. The author has contributed to research in topics: Autophagy & Phagosome. The author has an hindex of 101, co-authored 269 publications receiving 45639 citations. Previous affiliations of Vojo Deretic include University of Texas at Austin & University of Michigan.
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Patent
Diagnosis of infection in the lungs of patients
Graham S. Timmins,Vojo Deretic +1 more
TL;DR: In this paper, a method for detecting P aeruginosa infection and bacterial burden in the lungs of patients who are at risk for P. aerugINOSA infections, especially including patients with Cystic Fibrosis (CF), was proposed.
Book ChapterDOI
Signal Transduction and Environmental Stress in Control of Pseudomonas Aeruginosa Virulence
TL;DR: Pseudomonas aeruginosa causes life threatening infections in several classes of patients with compromised natural defense systems, among notorious examples are the systemic infections in severely burned patients and chronic respiratory infections in individuals with cystic fibrosis.
Journal ArticleDOI
Atg8 family proteins, LIR/AIM motifs and other interaction modes
Vladimir V. Rogov,Ioannis P. Nezis,Panagiotis Tsapras,Hong-Jiang Zhang,Yasin F. Dagdas,Nobuo N. Noda,Hitoshi Nakatogawa,Martina Wirth,Stephane Mouilleron,David G. McEwan,Christian Behrends,Vojo Deretic,Zvulun Elazar,Sharon A. Tooze,Ivan Dikic,Trond Lamark,Terje Johansen +16 more
TL;DR: The Atg8 family of ubiquitin-like proteins play pivotal roles in autophagy and other processes involving vesicle fusion and transport where the lysosome/ vacuole is the end station as discussed by the authors .
Journal ArticleDOI
Faculty Opinions recommendation of Parallel phospholipid transfer by Vps13 and Atg2 determines autophagosome biogenesis dynamics.
Posted ContentDOI
Optical induction of autophagy via Transcription factor EB (TFEB) reduces pathological tau in neurons
TL;DR: It is shown light-induced TFEB has the capacity to reduce p-Tau burden in AD patient-derived human iPSC-neurons and suggests that optically-regulatable gene expression of T FEB unlocks opto-therapeutics to treat AD and other dementias.