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William A. Blattner

Researcher at University of Maryland, Baltimore

Publications -  345
Citations -  26953

William A. Blattner is an academic researcher from University of Maryland, Baltimore. The author has contributed to research in topics: Population & Virus. The author has an hindex of 80, co-authored 345 publications receiving 26023 citations. Previous affiliations of William A. Blattner include RTI International & Boston University.

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Cytogenetic studies in human T-cell lymphoma virus (HTLV)-positive leukemia-lymphoma in the United States.

TL;DR: The characteristic clinical features of these 6 patients were aggressive course, short survival, poor response to chemotherapy, high white blood cell counts, hypercalcemia, and bone lesions, whereas cytogenetically abnormal patients without chromosome 6q deletion tended to have a more indolent course.
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Malignant mesenchymoma and birth defects. Prenatal exposure to phenytoin.

TL;DR: Following combination chemotherapy for metastases, the patient experienced a 7-year disease-free interval, which is consistent with recent improvement in the treatment of soft-tissue sarcomas.
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Rapid Clearance of Virus after Acute HIV-1 Infection: Correlates of Risk of AIDS

TL;DR: Among a subset of patients, rapid clearance of plasma HIV-1 after peak viremia is associated with lower viral set point, prolonged virus suppression before loss of virologic control, and decreased risk of AIDS.
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Concordance of Human Leukocyte Antigen Haplotype-Sharing, CD4 Decline and AIDS in Hemophilic Siblings. Multicenter Hemophilia Cohort and Hemophilia Growth and Development Studies

TL;DR: Sibling pairs sharing one or two haplotypes were significantly concordant in CD4 decline and AIDS status within 5 years of seroconversion, demonstrating that HIV‐1 disease progression is associated with the genes in the major histocompatibility complex that regulate the host's immune response.
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A survey of the human T-cell lymphotropic virus type I (HTLV-I) in south-western Colombia.

TL;DR: A hypothesis emerging from this study is that the lower socio‐economic status of lowland residents and associated diseases, particularly untreated syphilis and other sexually transmitted diseases, may explain the increased HTLV‐I seropositivity rates in this population.