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William Eisner
Researcher at Public Health Research Institute
Publications - 17
Citations - 2703
William Eisner is an academic researcher from Public Health Research Institute. The author has contributed to research in topics: Methicillin-resistant Staphylococcus aureus & Staphylococcus aureus. The author has an hindex of 16, co-authored 17 publications receiving 2647 citations. Previous affiliations of William Eisner include Michigan Technological University & University of Minnesota.
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Journal ArticleDOI
Role of External Bacterial Flora in the Pathogenesis of Acute Postoperative Endophthalmitis
Mark G. Speaker,Florence A. Milch,Mahendra Shah,William Eisner,Barry N. Kreiswirth,Barry N. Kreiswirth +5 more
TL;DR: Using techniques of molecular epidemiology, the authors demonstrate that an organism isolated from the vitreous was genetically indistinguishable from an isolate recovered from the patient's eyelid, conjunctiva, or nose in 14 (82%) of 17 cases of endophthalmitis.
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Evidence for a Clonal Origin of Methicillin Resistance in Staphylococcus aureus
Barry N. Kreiswirth,John Kornblum,John Kornblum,Robert D. Arbeit,William Eisner,Joel N. Maslow,Allison McGeer,Donald E. Low,Richard P. Novick +8 more
TL;DR: An investigation of the clonality of 472 isolates by DNA hybridization was performed, suggesting that mecA divergence preceded the acquisition of Tn554 in all cases and therefore thatmecA may have been acquired just once by S. aureus.
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Origin and interstate spread of a New York City multidrug-resistant Mycobacterium tuberculosis clone family
Pablo Bifani,Bonnie B. Plikaytis,Vivek Kapur,Vivek Kapur,Kathryn E. Stockbauer,Xi Pan,Michael L. Lutfey,Soraya L. Moghazeh,William Eisner,Thomas M. Daniel,Mark H. Kaplan,Jack T. Crawford,James M. Musser,Barry N. Kreiswirth +13 more
TL;DR: The results document the molecular origin and spread of progeny of a closely related family of multidrug-resistant M tuberculosis strains that have recently shared a common ancestor and undergone clonal expansion.
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Emergence of vancomycin-resistant enterococci in New York City
Thomas R. Frieden,Sonal S. Munsiff,G. Williams,Y. Faur,Barry N. Kreiswirth,Don E. Low,Barbara M. Willey,S. Warren,William Eisner +8 more
TL;DR: Molecular analyses suggest that a highly mobile genetic element--eg, a transposon--is responsible for the rapid spread of vancomycin resistance in New York City.
Journal ArticleDOI
Molecular Epidemiology of Methicillin-Resistant Staphylococcus aureus in 12 New York Hospitals
Richard B. Roberts,Alexandre de Lencastre,William Eisner,Elena P. Severina,Bo Shopsin,Barry N. Kreiswirth,Alexander Tomasz +6 more
TL;DR: Using both mecA:Tn554 probes and PFGE, MRSA clusters and outbreaks may be detected and provide a rationale for appropriate infection control intervention.