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Wolfgang Völkel

Researcher at University of Würzburg

Publications -  89
Citations -  7413

Wolfgang Völkel is an academic researcher from University of Würzburg. The author has contributed to research in topics: Phthalate & Tolerable daily intake. The author has an hindex of 42, co-authored 84 publications receiving 6534 citations. Previous affiliations of Wolfgang Völkel include Lawrence Livermore National Laboratory & Ludwig Maximilian University of Munich.

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Metabolism and kinetics of bisphenol a in humans at low doses following oral administration.

TL;DR: The efficient glucuronidation of bisphenol A and the rapid excretion of the formed glucuronide result in a low body burden of the estrogenic bispenol A in humans following oral absorption of low doses.
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Perfluorinated compounds--exposure assessment for the general population in Western countries.

TL;DR: Overall, the contribution of PFOS and PFOA precursors to total exposure seems to be limited, and besides this background exposure of the general population, a specific additional exposure may occur which causes an increased PFC body burden.
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Human exposure to bisphenol A by biomonitoring: Methods, results and assessment of environmental exposures

TL;DR: In urine samples from several cohorts, bisphenol A (as glucuronide) was present in average concentrations in the range of 1-3 microg/L suggesting that daily human exposure to bispenol A is below 6 microg per person for the majority of the population.
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Pre- and postnatal exposure to perfluorinated compounds (PFCs).

TL;DR: Low levels of PFCs in cord sera and an increase in concentrations through the first months of infant life are found and this intake led to a body burden at the age of six months similar to (PFOS) or higher than (PFOA) that found in adults.
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Critical evaluation of key evidence on the human health hazards of exposure to bisphenol A

TL;DR: The Advisory Committee of the German Society of Toxicology reviewed the background and cutting-edge topics of this BPA controversy and concluded that rodent data can well be used as a basis for human risk evaluation and conjectures that rats are insensitive to estrogens compared to humans can be refuted.