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Xiongjie Shi
Researcher at Wuhan University
Publications - 49
Citations - 3697
Xiongjie Shi is an academic researcher from Wuhan University. The author has contributed to research in topics: Developmental toxicity & Zebrafish. The author has an hindex of 25, co-authored 43 publications receiving 2894 citations. Previous affiliations of Xiongjie Shi include Sun Yat-sen University & Chinese Academy of Sciences.
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Journal ArticleDOI
Developmental toxicity and alteration of gene expression in zebrafish embryos exposed to PFOS.
TL;DR: The overall results indicated that zebrafish embryos constitute a reliable model for testing the developmental toxicity of PFOS, and the gene expression patterns in the embryos were able to reveal some potential mechanisms of developmental toxicity.
Journal ArticleDOI
Perfluorinated compounds in the Pearl River and Yangtze River of China.
M.K. So,Yuichi Miyake,W. Y. Yeung,Y. M. Ho,Sachi Taniyasu,Pawel Rostkowski,Pawel Rostkowski,Nobuyoshi Yamashita,Bingsheng Zhou,Xiongjie Shi,Jingzhi Wang,John P. Giesy,John P. Giesy,John P. Giesy,Hui Yu,Paul K.S. Lam +15 more
TL;DR: Generally, PFOS was the dominant PFC found in samples from the Pearl River, while PFOA was the predominant PFC in water from the Yangtze River, which indicates the presence of dissimilar sources in these two regions.
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Induction of oxidative stress and apoptosis by PFOS and PFOA in primary cultured hepatocytes of freshwater tilapia (Oreochromis niloticus)
TL;DR: The overall results demonstrated that PFOS and PFOA are able to produce oxidative stress and induce apoptosis with involvement of caspases in primary cultured tilapia hepatocytes.
Journal ArticleDOI
The role of Nrf2 and MAPK pathways in PFOS-induced oxidative stress in zebrafish embryos
Xiongjie Shi,Bingsheng Zhou +1 more
TL;DR: It is found that coexposure with sulforaphane, an Nrf2 activator, could significantly protect against PFOS-induced ROS generation, whereas inhibition of MAPKs did not exhibit significant effects on PFos-induced HO-1 gene expression and ROS production.
Journal ArticleDOI
Hexabromocyclododecane-induced developmental toxicity and apoptosis in zebrafish embryos
Jun Deng,Liqin Yu,Chunsheng Liu,Ke Yu,Xiongjie Shi,Leo W. Y. Yeung,Paul K.S. Lam,Rudolf S.S. Wu,Bingsheng Zhou +8 more
TL;DR: The overall results demonstrate that waterborne HBCD is able to produce oxidative stress and induce apoptosis through the involvement of caspases in zebrafish embryos, and indicates that zebra fish embryos can serve as a reliable model for the developmental toxicity of H BCD.