Y
Yahong Zhang
Researcher at Henan University
Publications - 31
Citations - 461
Yahong Zhang is an academic researcher from Henan University. The author has contributed to research in topics: Apoptosis & Engineering. The author has an hindex of 11, co-authored 23 publications receiving 363 citations. Previous affiliations of Yahong Zhang include Shenyang Pharmaceutical University.
Papers
More filters
Journal ArticleDOI
Reactive oxygen species contribute to oridonin-induced apoptosis and autophagy in human cervical carcinoma HeLa cells.
TL;DR: ROS plays a critical role in oridonin-induced apoptosis and autophagy in HeLa cells and was significantly reduced in the presence of the ROS scavenger NAC, while the oridinin- induced ROS generation was markedly reduced.
Journal ArticleDOI
NF-κb facilitates oridonin-induced apoptosis and autophagy in HT1080 cells through a p53-mediated pathway
TL;DR: NF-kappaB promotes oridonin-induced apoptotic and autophagic cell death through regulating p53 activation in HT1080 cells, demonstrating that they act in synergy to mediate cell death.
Journal ArticleDOI
Fas-mediated autophagy requires JNK activation in HeLa cells.
Yahong Zhang,Yingliang Wu,Yan Cheng,Zhenkun Zhao,Shin-ichi Tashiro,Satoshi Onodera,Takashi Ikejima +6 more
TL;DR: It is suggested that JNK, but not caspase 8, involves in Fas-mediated CH11-induced autophagy in HeLa cells, and this Autophagy plays a protective role in CH 11-induced cell death.
Journal ArticleDOI
Involvement of PKC signal pathways in oridonin-induced autophagy in HeLa cells: a protective mechanism against apoptosis.
TL;DR: It is demonstrated that PKC enhances oridonin-induced autophagy against apoptosis through regulating its downstream factors Raf-1 and JNK in HeLa cells.
Journal ArticleDOI
Coxsackievirus A6 Induces Cell Cycle Arrest in G0/G1 Phase for Viral Production.
Zengyan Wang,Yue Wang,Shaohua Wang,Xiangling Meng,Fengmei Song,Wenbo Huo,Shuxia Zhang,Junliang Chang,Jingliang Li,Baisong Zheng,Yanqiu Liu,Yahong Zhang,Wenyan Zhang,Jinghua Yu +13 more
TL;DR: It is demonstrated for the first time that CVA6 infection arrests the host cell cycle in G0/G1-phase via non-structural proteins 3D and 3C, which may provide favorable environments for virus production.