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Yasuhisa Nogi

Researcher at Saitama Medical University

Publications -  29
Citations -  1137

Yasuhisa Nogi is an academic researcher from Saitama Medical University. The author has contributed to research in topics: RNA polymerase I & RNA polymerase. The author has an hindex of 15, co-authored 29 publications receiving 1076 citations.

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Journal ArticleDOI

Essential Role of p38 Mitogen-activated Protein Kinase in Cathepsin K Gene Expression during Osteoclastogenesis through Association of NFATc1 and PU.1 *

TL;DR: The results suggest that the RANKL-induced cathepsin K gene expression is cooperatively regulated by the combination of the transcription factors and p38 MAP kinase in a gradual manner.
Journal Article

Cancer-associated Myofibroblasts Possess Various Factors to Promote Endometrial Tumor Progression

TL;DR: It is indicated that the majority of the myofibroblasts as well as cancer epithelial cells are proliferating because of their positive staining of proliferating cell nuclear antigen and Ki-67, suggesting directly that the my ofibro Blasts might participate in the progression of tumor cells in terms of cancer cell growth stimulation and also activated initiation of angiogenesis.
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Molecular cloning of ring finger protein 21 (RNF21)/interferon-responsive finger protein (ifp1), which possesses two RING-B box-coiled coil domains in tandem.

TL;DR: Findings suggest that RNF21 is a downstream gene that may mediate interferon's biological action, and that it possesses at least three kinds of isoforms, due to alternative splicing, consisting of the long form with the R BCC-RBCC-B30.2 domain, and the short form with only the RBCC domain.
Journal ArticleDOI

Two RNA Polymerase I Subunits Control the Binding and Release of Rrn3 during Transcription

TL;DR: A dual role of the Rpa49-Rpa34 dimer during the recruitment of Rrn3 and its subsequent dissociation from the elongating polymerase is suggested.
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Regulation of expression of the galactose gene cluster in Saccharomyces cerevisiae. II. The isolation and dosage effect of the regulatory gene GAL80.

TL;DR: It is suggested that the intracellular inducer interacts with the GAL80 product and that GAL 80 molecules directly bind the G AL cluster genes with an affinity different from one gene to another.