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Yehezkel Ben-Ari
Researcher at French Institute of Health and Medical Research
Publications - 462
Citations - 46449
Yehezkel Ben-Ari is an academic researcher from French Institute of Health and Medical Research. The author has contributed to research in topics: Hippocampal formation & Excitatory postsynaptic potential. The author has an hindex of 110, co-authored 459 publications receiving 44293 citations. Previous affiliations of Yehezkel Ben-Ari include Aix-Marseille University & Medical Research Council.
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Inhibitory actions of the gamma‐aminobutyric acid in pediatric Sturge‐Weber syndrome
Roman Tyzio,Ilgam Khalilov,Alfonso Represa,Valérie Crépel,Yuri Zilberter,Sylvain Rheims,Laurent Aniksztejn,Rosa Cossart,Romain Nardou,Marat Mukhtarov,Marat Minlebaev,Jérôme Epsztein,Mathieu Milh,Hélène Becq,Isabel Jorquera,Christine Bulteau,Martine Fohlen,Viviana Oliver,Olivier Dulac,Georg Dorfmüller,Olivier Delalande,Yehezkel Ben-Ari,Roustem Khazipov +22 more
TL;DR: The properties of neurons from human pediatric Sturge‐Weber syndrome cortex in vitro are explored and whether gamma‐aminobutyric acid (GABA) excites neurons in SWS cortex, as has been suggested for various types of epilepsies is tested.
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The K+ channel opener diazoxide enhances glutamatergic currents and reduces GABAergic currents in hippocampal neurons.
TL;DR: It is concluded that, in the hippocampus, diazoxide enhances the excitatory glutamatergic currents and reduces the GABAergic inhibition, thus generating paroxystic activities and it is suggested that these effects are mediated by second messenger cascades.
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Nature and nurture in brain development
TL;DR: This meeting provided stipends to enable participation of young scientists from less developed countries and is a major aspect of these meetings that the organisers hope to maintain.
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Exclusively inhibitory action of iontophoretic acetylcholine on single neurones of feline thalamus.
TL;DR: The nucleus reticularis of the cat thalamus may well prove to be an appropriate area in which to study the depressant action of A Ch on central mammalian neurones since almost every cell encountered proved to be inhibited by a short, low amplitude pulse of ACh.
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GABA regulates stem cell proliferation before nervous system formation.
TL;DR: It is shown that autocrine/paracrine γ-aminobutyric acid (GABA) signalling by means of GABAA receptors negatively controls ES cell and peripheral neural crest stem (NCS) cell proliferation, preimplantation embryonic growth and proliferation in the boundary-cap stemcell niche, resulting in an attenuation of neuronal progenies from this stem cell niche.