scispace - formally typeset
Y

Yingpeng Xie

Researcher at City University of Hong Kong

Publications -  19
Citations -  445

Yingpeng Xie is an academic researcher from City University of Hong Kong. The author has contributed to research in topics: Pseudomonas syringae & Virulence. The author has an hindex of 8, co-authored 15 publications receiving 235 citations. Previous affiliations of Yingpeng Xie include Nankai University.

Papers
More filters
Journal ArticleDOI

An integrated genomic regulatory network of virulence-related transcriptional factors in Pseudomonas aeruginosa

TL;DR: A genome-wide, network-based approach to dissect the crosstalk between 20 key virulence-related TFs led to the identification of novel functions for two TFs (ExsA and GacA) in quorum sensing and nitrogen metabolism.
Journal ArticleDOI

Pleiotropic Effects of c-di-GMP Content in Pseudomonas syringae.

TL;DR: It is demonstrated that c-di-GMP regulates the virulence and stress response in P. syringae, which suggests that tuning its level could be a new strategy to protect plants from attacks by this pathogen.
Journal ArticleDOI

RNA G-Quadruplex Structures Mediate Gene Regulation in Bacteria.

TL;DR: This study uncovers important regulatory functions of rG4s in bacterial pathogenicity and metabolic pathways and strongly suggests that rG 4s exist and can be detected in a wide range of bacterial species.
Journal ArticleDOI

RpoN-Dependent Direct Regulation of Quorum Sensing and the Type VI Secretion System in Pseudomonas aeruginosa PAO1.

TL;DR: This work shows that RpoN plays critical and global roles in the regulation of bacterial pathogenicity and fitness and the functional characterization of RPON-mediated regulation will improve the current understanding of the regulatory network of quorum sensing and virulence in P. aeruginosa and other bacteria.
Journal ArticleDOI

Regulation of type III secretion system in Pseudomonas syringae.

TL;DR: Overall, these studies have suggested the existence of a highly intricate regulatory network for T3SS, which thus also controls the pathogenicity of P. syringae.