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Young Jun Kim

Researcher at Korea University

Publications -  261
Citations -  6681

Young Jun Kim is an academic researcher from Korea University. The author has contributed to research in topics: Conjugated linoleic acid & Oxidative stress. The author has an hindex of 36, co-authored 233 publications receiving 5498 citations. Previous affiliations of Young Jun Kim include Food and Drug Administration & Cornell University.

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Quantification of polyphenolics and their antioxidant capacity in fresh plums.

TL;DR: A positive relationship (correlation coefficient r (2)() = 0.977) was presented between total phenolics and VCEAC, suggesting polyphenolics would play an important role in free radical scavenging, and Beltsville Elite B70197 showed the highest amounts of total phenolic and total flavonoids and the highest VCEac.
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Sweet and sour cherry phenolics and their protective effects on neuronal cells.

TL;DR: Overall results showed that cherries are rich in phenolics, especially in anthocyanins, with a strong antineurodegenerative activity and that they can serve as a good source of biofunctional phytochemicals in the authors' diet.
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Epigallocatechin-3-gallate, a histone acetyltransferase inhibitor, inhibits EBV-induced B lymphocyte transformation via suppression of RelA acetylation.

TL;DR: In this paper, epigallocatechin-3-gallate (EGCG) was identified as a histone acetyltransferase inhibitor (HATi) with global specificity for the majority of HAT enzymes but with no activity toward epigenetic enzymes including HDAC, SIRT1, and HMTase.
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The enrichment of a ruminal bacterium (Megasphaera elsdenii YJ-4) that produces the trans-10, cis-12 isomer of conjugated linoleic acid.

TL;DR: Aims: To isolate predominant ruminal bacteria that produce trans‐10, cis‐12 conjugated linoleic acid (CLA) from linoleics acid (LA).
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Effect of linoleic acid concentration on conjugated linoleic acid production by Butyrivibrio fibrisolvens A38

TL;DR: Growing cultures of Butyrivibrio fibrisolvens A38 inocula were inhibited by as little as 15 μM linoleic acid (LA), but growing cultures tolerated 10-fold more LA before growth was inhibited, and the flow of CLA from the rumen may be due to LA-dependent bacterial inactivation, death, or lysis.