Example of Neuropathology and Applied Neurobiology format
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Example of Neuropathology and Applied Neurobiology format Example of Neuropathology and Applied Neurobiology format Example of Neuropathology and Applied Neurobiology format Example of Neuropathology and Applied Neurobiology format Example of Neuropathology and Applied Neurobiology format Example of Neuropathology and Applied Neurobiology format
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Example of Neuropathology and Applied Neurobiology format Example of Neuropathology and Applied Neurobiology format Example of Neuropathology and Applied Neurobiology format Example of Neuropathology and Applied Neurobiology format Example of Neuropathology and Applied Neurobiology format Example of Neuropathology and Applied Neurobiology format
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Neuropathology and Applied Neurobiology — Template for authors

Publisher: Wiley
Categories Rank Trend in last 3 yrs
Pathology and Forensic Medicine #5 of 191 up up by 2 ranks
Neurology #5 of 156 up up by 6 ranks
Neurology (clinical) #15 of 343 up up by 8 ranks
Histology #4 of 60 down down by 1 rank
Physiology (medical) #9 of 98 down down by 2 ranks
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 177 Published Papers | 2035 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 03/06/2020
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Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

7.5

9% from 2018

Impact factor for Neuropathology and Applied Neurobiology from 2016 - 2019
Year Value
2019 7.5
2018 6.878
2017 6.059
2016 5.347
graph view Graph view
table view Table view

11.5

4% from 2019

CiteRatio for Neuropathology and Applied Neurobiology from 2016 - 2020
Year Value
2020 11.5
2019 11.1
2018 11.0
2017 9.9
2016 9.1
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 9% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 4% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

2.538

2% from 2019

SJR for Neuropathology and Applied Neurobiology from 2016 - 2020
Year Value
2020 2.538
2019 2.497
2018 2.42
2017 2.236
2016 2.12
graph view Graph view
table view Table view

1.696

4% from 2019

SNIP for Neuropathology and Applied Neurobiology from 2016 - 2020
Year Value
2020 1.696
2019 1.627
2018 1.272
2017 1.128
2016 1.137
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has increased by 2% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 4% in last years.
  • This journal’s SNIP is in the top 10 percentile category.
Neuropathology and Applied Neurobiology

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Wiley

Neuropathology and Applied Neurobiology

Neuropathology and Applied Neurobiology is an international journal, sponsored by the British Neuropathological Society, for the publication of original papers and authoritative reviews on the disease mechanisms and the clinical pathology of disorders of brain, nerve and muscl...... Read More

Medicine

i
Last updated on
03 Jun 2020
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ISSN
0305-1846
i
Impact Factor
High - 1.178
i
Open Access
Yes
i
Sherpa RoMEO Archiving Policy
Yellow faq
i
Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Bibliography Name
apa
i
Citation Type
Numbered
[25]
i
Bibliography Example
Beenakker, C.W.J. (2006) Specular andreev reflection in graphene.Phys. Rev. Lett., 97 (6), 067 007. URL 10.1103/PhysRevLett.97.067007.

Top papers written in this journal

Journal Article DOI: 10.1111/NAN.12011
Review: Activation patterns of microglia and their identification in the human brain
Delphine Boche1, Victor Hugh Perry1, James A. R. Nicoll2, James A. R. Nicoll1

Abstract:

Microglia in the central nervous system are usually maintained in a quiescent state. When activated, they can perform many diverse functions which may be either beneficial or harmful depending on the situation. Although microglial activation may be accompanied by changes in morphology, morphological changes cannot accurately ... Microglia in the central nervous system are usually maintained in a quiescent state. When activated, they can perform many diverse functions which may be either beneficial or harmful depending on the situation. Although microglial activation may be accompanied by changes in morphology, morphological changes cannot accurately predict the function being undertaken by a microglial cell. Studies of peripheral macrophages and in vitro and animal studies of microglia have resulted in the definition of specific activation states: M1 (classical activation) and M2 (sometimes subdivided into alternative activation and acquired deactivation). Some authors have suggested that these might be an overlapping continuum of functions rather than discrete categories. In this review, we consider translational aspects of our knowledge of microglia: specifically, we discuss the question as to what extent different activation states of microglia exist in the human central nervous system, which tools can be used to identify them and emerging evidence for such changes in ageing and in Alzheimer's disease. read more read less

Topics:

Neuroinflammation (58%)58% related to the paper
806 Citations
open accessOpen access Journal Article DOI: 10.1111/J.1365-2990.2007.00874.X
Parkinson's disease: a dual-hit hypothesis.
Christopher Hawkes1, K. Del Tredici2, Heiko Braak2

Abstract:

Accumulating evidence suggests that sporadic Parkinson's disease has a long prodromal period during which several non-motor features develop, in particular, impairment of olfaction, vagal dysfunction and sleep disorder. Early sites of Lewy pathology are the olfactory bulb and enteric plexus of the stomach. We propose that a n... Accumulating evidence suggests that sporadic Parkinson's disease has a long prodromal period during which several non-motor features develop, in particular, impairment of olfaction, vagal dysfunction and sleep disorder. Early sites of Lewy pathology are the olfactory bulb and enteric plexus of the stomach. We propose that a neurotropic pathogen, probably viral, enters the brain via two routes: (i) nasal, with anterograde progression into the temporal lobe; and (ii) gastric, secondary to swallowing of nasal secretions in saliva. These secretions might contain a neurotropic pathogen that, after penetration of the epithelial lining, could enter axons of the Meissner's plexus and, via transsynaptic transmission, reach the preganglionic parasympathetic motor neurones of the vagus nerve. This would allow retrograde transport into the medulla and, from here, into the pons and midbrain until the substantia nigra is reached and typical aspects of disease commence. Evidence for this theory from the perspective of olfactory and autonomic dysfunction is reviewed, and the possible routes of pathogenic invasion are considered. It is concluded that the most parsimonious explanation for the initial events of sporadic Parkinson's disease is pathogenic access to the brain through the stomach and nose - hence the term 'dual-hit'. read more read less

Topics:

Parkinson's disease (54%)54% related to the paper, Olfactory bulb (51%)51% related to the paper, Substantia nigra (51%)51% related to the paper, Olfaction (51%)51% related to the paper, Vagus nerve (50%)50% related to the paper
View PDF
775 Citations
open accessOpen access Journal Article DOI: 10.1111/J.1365-2990.2010.01139.X
Review: cerebral microvascular pathology in ageing and neurodegeneration.
William R. Brown1, Clara R. Thore

Abstract:

This review of age-related brain microvascular pathologies focuses on topics studied by this laboratory, including anatomy of the blood supply, tortuous vessels, venous collagenosis, capillary remnants, vascular density and microembolic brain injury. Our studies feature thick sections, large blocks embedded in celloidin, and ... This review of age-related brain microvascular pathologies focuses on topics studied by this laboratory, including anatomy of the blood supply, tortuous vessels, venous collagenosis, capillary remnants, vascular density and microembolic brain injury. Our studies feature thick sections, large blocks embedded in celloidin, and vascular staining by alkaline phosphatase. This permits study of the vascular network in three dimensions, and the differentiation of afferent from efferent vessels. Current evidence suggests that there is decreased vascular density in ageing, Alzheimer's disease and leukoaraiosis, and cerebrovascular dysfunction precedes and accompanies cognitive dysfunction and neurodegeneration. A decline in cerebrovascular angiogenesis may inhibit recovery from hypoxia-induced capillary loss. Cerebral blood flow is inhibited by tortuous arterioles and deposition of excessive collagen in veins and venules. Misery perfusion due to capillary loss appears to occur before cell loss in leukoaraiosis, and cerebral blood flow is also reduced in the normal-appearing white matter. Hypoperfusion occurs early in Alzheimer's disease, inducing white matter lesions and correlating with dementia. In vascular dementia, cholinergic reductions are correlated with cognitive impairment, and cholinesterase inhibitors have some benefit. Most lipid microemboli from cardiac surgery pass through the brain in a few days, but some remain for weeks. They can cause what appears to be a type of vascular dementia years after surgery. Donepezil has shown some benefit. Emboli, such as clots, cholesterol crystals and microspheres can be extruded through the walls of cerebral vessels, but there is no evidence yet that lipid emboli undergo such extravasation. read more read less

Topics:

Vascular dementia (59%)59% related to the paper, Microcirculation (55%)55% related to the paper, Cerebral blood flow (55%)55% related to the paper, Cerebral veins (52%)52% related to the paper, Leukoaraiosis (52%)52% related to the paper
View PDF
629 Citations
open accessOpen access Journal Article DOI: 10.1111/J.1365-2990.2012.01306.X
Review: Microglia of the aged brain: primed to be activated and resistant to regulation
Diana M. Norden1, Jonathan P. Godbout1

Abstract:

Innate immunity within the central nervous system (CNS) is primarily provided by resident microglia. Microglia are pivotal in immune surveillance and also facilitate the co-ordinated responses between the immune system and the brain. For example, microglia interpret and propagate inflammatory signals that are initiated in the... Innate immunity within the central nervous system (CNS) is primarily provided by resident microglia. Microglia are pivotal in immune surveillance and also facilitate the co-ordinated responses between the immune system and the brain. For example, microglia interpret and propagate inflammatory signals that are initiated in the periphery. This transient microglial activation helps mount the appropriate physiological and behavioural response following peripheral infection. With normal ageing, however, microglia develop a more inflammatory phenotype. For instance, in several models of ageing there are increased pro-inflammatory cytokines in the brain and increased expression of inflammatory receptors on microglia. This increased inflammatory status of microglia with ageing is referred to as primed, reactive or sensitized. A modest increase in the inflammatory profile of the CNS and altered microglial function in ageing has behavioural and cognitive consequences. Nonetheless, there are major differences in microglial biology between young and old age when the immune system is challenged and microglia are activated. In this context, microglial activation is amplified and prolonged in the aged brain compared with adults. The cause of this amplified microglial activation may be related to impairments in several key regulatory systems with age that make it more difficult to resolve microglial activation. The consequences of impaired regulation and microglial hyper-activation following immune challenge are exaggerated neuroinflammation, sickness behaviour, depressive-like behaviour and cognitive deficits. Therefore the purpose of this review is to discuss the current understanding of age-associated microglial priming, consequences of priming and reactivity, and the impairments in regulatory systems that may underlie these age-related deficits. read more read less

Topics:

Neuroinflammation (61%)61% related to the paper, Microglia (51%)51% related to the paper
View PDF
616 Citations
open accessOpen access Journal Article DOI: 10.1111/J.1365-2990.2007.00926.X
Solutes, but not cells, drain from the brain parenchyma along basement membranes of capillaries and arteries: significance for cerebral amyloid angiopathy and neuroimmunology

Abstract:

Elimination of interstitial fluid and solutes plays a role in homeostasis in the brain, but the pathways are unclear. Previous work suggests that interstitial fluid drains along the walls of arteries. Aims: to define the pathways within the walls of capillaries and arteries for drainage of fluid and solutes out of the brain. ... Elimination of interstitial fluid and solutes plays a role in homeostasis in the brain, but the pathways are unclear. Previous work suggests that interstitial fluid drains along the walls of arteries. Aims: to define the pathways within the walls of capillaries and arteries for drainage of fluid and solutes out of the brain. Methods: Fluorescent soluble tracers, dextran (3 kDa) and ovalbumin (40 kDa), and particulate fluospheres (0.02 μm and 1.0 μm in diameter) were injected into the corpus striatum of mice. Brains were examined from 5 min to 7 days by immunocytochemistry and confocal microscopy. Results: soluble tracers initially spread diffusely through brain parenchyma and then drain out of the brain along basement membranes of capillaries and arteries. Some tracer is taken up by vascular smooth muscle cells and by perivascular macrophages. No perivascular drainage was observed when dextran was injected into mouse brains following cardiac arrest. Fluospheres expand perivascular spaces between vessel walls and surrounding brain, are ingested by perivascular macrophages but do not appear to leave the brain even following an inflammatory challenge with lipopolysaccharide or kainate. Conclusions: capillary and artery basement membranes act as ‘lymphatics of the brain’ for drainage of fluid and solutes; such drainage appears to require continued cardiac output as it ceases following cardiac arrest. This drainage pathway does not permit migration of cells from brain parenchyma to the periphery. Amyloid-β is deposited in basement membrane drainage pathways in cerebral amyloid angiopathy, and may impede elimination of amyloid-β and interstitial fluid from the brain in Alzheimer's disease. Soluble antigens, but not cells, drain from the brain by perivascular pathways. This atypical pattern of drainage may contribute to partial immune privilege of the brain and play a role in neuroimmunological diseases such as multiple sclerosis. read more read less

Topics:

Perivascular space (58%)58% related to the paper, Glymphatic system (56%)56% related to the paper, Cerebral arteries (53%)53% related to the paper, Interstitial fluid (53%)53% related to the paper, Parenchyma (51%)51% related to the paper
View PDF
518 Citations
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Frequently asked questions

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Yes, the template is compliant with the Neuropathology and Applied Neurobiology guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Neuropathology and Applied Neurobiology?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Neuropathology and Applied Neurobiology citation style.

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Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Neuropathology and Applied Neurobiology.

5. Can I use a manuscript in Neuropathology and Applied Neurobiology that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Neuropathology and Applied Neurobiology that you can download at the end.

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12. Is Neuropathology and Applied Neurobiology's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Neuropathology and Applied Neurobiology?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Neuropathology and Applied Neurobiology. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Neuropathology and Applied Neurobiology?

The 5 most common citation types in order of usage for Neuropathology and Applied Neurobiology are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

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16. Can I download Neuropathology and Applied Neurobiology in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Neuropathology and Applied Neurobiology Endnote style according to Elsevier guidelines.

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