Example of Current Rheumatology Reviews format
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Example of Current Rheumatology Reviews format Example of Current Rheumatology Reviews format Example of Current Rheumatology Reviews format Example of Current Rheumatology Reviews format Example of Current Rheumatology Reviews format Example of Current Rheumatology Reviews format Example of Current Rheumatology Reviews format Example of Current Rheumatology Reviews format
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Example of Current Rheumatology Reviews format Example of Current Rheumatology Reviews format Example of Current Rheumatology Reviews format Example of Current Rheumatology Reviews format Example of Current Rheumatology Reviews format Example of Current Rheumatology Reviews format Example of Current Rheumatology Reviews format Example of Current Rheumatology Reviews format
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open access Open Access ISSN: 15733971

Current Rheumatology Reviews — Template for authors

Publisher: Bentham Science
Categories Rank Trend in last 3 yrs
Rheumatology #35 of 56 down down by 2 ranks
journal-quality-icon Journal quality:
Medium
calendar-icon Last 4 years overview: 164 Published Papers | 400 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 19/06/2020
Insights & related journals
General info
Top papers
Popular templates
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FAQ

Journal Performance & Insights

  • CiteRatio
  • SJR
  • SNIP

CiteRatio is a measure of average citations received per peer-reviewed paper published in the journal.

2.4

CiteRatio for Current Rheumatology Reviews from 2016 - 2020
Year Value
2020 2.4
2019 2.4
2018 2.5
2017 2.1
2016 1.0
graph view Graph view
table view Table view

insights Insights

  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR) measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

0.492

2% from 2019

SJR for Current Rheumatology Reviews from 2016 - 2020
Year Value
2020 0.492
2019 0.484
2018 0.53
2017 0.586
2016 0.332
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has increased by 2% in last years.
  • This journal’s SJR is in the top 10 percentile category.

Source Normalized Impact per Paper (SNIP) measures actual citations received relative to citations expected for the journal's category.

0.629

8% from 2019

SNIP for Current Rheumatology Reviews from 2016 - 2020
Year Value
2020 0.629
2019 0.581
2018 0.75
2017 0.593
2016 0.302
graph view Graph view
table view Table view

insights Insights

  • SNIP of this journal has increased by 8% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Related Journals

open access Open Access ISSN: 1759720X e-ISSN: 17597218

SAGE

CiteRatio: 5.5 | SJR: 1.387 | SNIP: 2.262
open access Open Access ISSN: 15233774 e-ISSN: 15346307

Springer

CiteRatio: 6.6 | SJR: 1.393 | SNIP: 1.654
open access Open Access ISSN: 15460096

Springer

CiteRatio: 4.4 | SJR: 1.051 | SNIP: 1.315
open access Open Access ISSN: 34967 e-ISSN: 14682060
recommended Recommended

BMJ Publishing Group

CiteRatio: 28.7 | SJR: 6.333 | SNIP: 4.294

Current Rheumatology Reviews

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Bentham Science

Current Rheumatology Reviews

Approved by publishing and review experts on SciSpace, this template is built as per for Current Rheumatology Reviews formatting guidelines as mentioned in Bentham Science author instructions. The current version was created on 19 Jun 2020 and has been used by 532 authors to write and format their manuscripts to this journal.

Rheumatology

Medicine

i
Last updated on
19 Jun 2020
i
ISSN
1573-3971
i
Impact Factor
Low - 0.098
i
Open Access
No
i
Sherpa RoMEO Archiving Policy
Yellow faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
Vancouver
i
Citation Type
Numbered
[25]
i
Bibliography Example
Blonder, G E, Tinkham, M, & Klapwijk, T M. Transition from metallic to tunnel- ing regimes in superconducting microconstrictions: Excess current, charge imbalance, and supercurrent conversion. Phys. Rev. B. 2013;87(10):100510.

Top papers written in this journal

Journal Article DOI: 10.2174/1573397112666151231110521
Irritable Bowel Syndrome: A Clinical Review
Michael D. Cashman1, Daniel K. Martin, Sonu Dhillon, Srinivas R. Puli

Abstract:

Symptoms of irritable bowel syndrome (IBS) are common in population studies including chronic abdominal pain associated with altered bowel habits. Patients often have associated gastrointestinal and somatic symptoms suggesting a possible common contributing mechanism, but the heterogeneous symptom patterns of individual patie... Symptoms of irritable bowel syndrome (IBS) are common in population studies including chronic abdominal pain associated with altered bowel habits. Patients often have associated gastrointestinal and somatic symptoms suggesting a possible common contributing mechanism, but the heterogeneous symptom patterns of individual patients make generalizations difficult. The pathophysiology of IBS is incompletely understood but includes disturbances of the brain-gut axis. Central mechanisms are: the psychosocial history and environment, dysfunctional brain processing of peripheral signals attributed to the intestine including the enteric nervous system, the microbiome and the innate and adaptive immune system. As a result there is visceral hypersensitivity and disturbed intestinal secretory and motor activity. Some mechanisms of visceral pain hypersensitivity may overlap with other pain syndromes including fibromyalgia (FMS). Central Sensitization (CS) would offer a way to conceptualize an integration of life experience and psychologic response into a biopsychosocial framework of pathophysiology, diagnosis and treatment of IBS. Corticotropin-releasing factor, a principle regulator in the stress and pain response may contribute to a neuroendocrine mechanism for the brain-gut interaction. The positive diagnostic approach to IBS symptoms to avoid excess testing and enhance the patient-provider therapeutic relationship requires the recognition of the "cluster" of IBS symptoms while identifying "alarm" symptoms requiring specific attention. The severity of the symptoms and other individual psychosocial factors characterize patients who seek medical care. The presence of significant psychosocial comorbidities adds to the complexity of management which often requires a multidisciplinary approach. Several treatment options exist but no single method is effective for all the symptoms of IBS. The therapeutic benefit of the well-executed physician-patient relationship is considered essential to success in managing IBS symptoms over the long term. read more read less

Topics:

Irritable bowel syndrome (57%)57% related to the paper, Visceral pain (52%)52% related to the paper, Fibromyalgia (52%)52% related to the paper, Population (51%)51% related to the paper
227 Citations
Journal Article DOI: 10.2174/157339711102150702112236
Editorial review: an update on central sensitivity syndromes and the issues of nosology and psychobiology.

Abstract:

Central sensitization (CS), simply defined as an amplified response of the central nervous system to peripheral input, is a concept of great importance in clinical medicine. It has helped to explain aspects of the pathophysiology of common diseases, e.g. fibromyalgia syndrome (FMS), irritable bowel syndrome, vulvodynia, heada... Central sensitization (CS), simply defined as an amplified response of the central nervous system to peripheral input, is a concept of great importance in clinical medicine. It has helped to explain aspects of the pathophysiology of common diseases, e.g. fibromyalgia syndrome (FMS), irritable bowel syndrome, vulvodynia, headaches, chronic pelvic pain and other overlapping conditions (collectively called central sensitivity syndromes, or CSS). It also applies to pain of complex regional pain syndrome, osteoarthritis (OA), rheumatoid arthritis (RA) and post-operative pain. The pathology-pain gap in CSS is readily explained by CS. Many FMS and other CSS patients have peripheral pathology, e.g. nociceptive areas in the muscles, arthritis, small fiber neuropathy and inflammation. Pro-inflammatory cytokines are elevated in some patients. Identification of CS in patients with structural pathology, e.g. OA and RA, has helped to explain why not all patients benefit from nonsteroidal anti-inflammatory drugs or joint replacement surgery, and require therapy directed at CS. Glial cells are important in pain processing. Remarkable advances have been achieved in neuroimaging, including visualization of grey matter and white matter, not only during provoked pain but also pain at rest. Based on CS mechanisms, targeted individual therapy may now be possible. Appropriate nosology is important particularly for effective patient care. Dichotomy of neurochemical-structural ("functional") and structural ("organic") pathology should be abandoned; many patients have both. Psychobiology is also biology. Patient-blaming terms like somatization, somatizer and catastrophizing should be avoided. For therapy, both pharmacological and non- pharmacological approaches are important, including recognition of subgroups and person/patient-centered care. read more read less

Topics:

Pelvic pain (57%)57% related to the paper, Complex regional pain syndrome (56%)56% related to the paper, Vulvodynia (52%)52% related to the paper, Irritable bowel syndrome (50%)50% related to the paper
124 Citations
open accessOpen access Journal Article DOI: 10.2174/157339711794474620
Choice of Biologic Therapy for Patients with Rheumatoid Arthritis: The Infection Perspective
Filip De Keyser1

Abstract:

Biologicals revolutionized the treatment of Rheumatoid Arthritis (RA). The targeted suppression of key inflammatory pathways involved in joint inflammation and destruction allows better disease control, which, however, comes at the price of an elevated infection risk due to relative immunosuppression. The disease-related infe... Biologicals revolutionized the treatment of Rheumatoid Arthritis (RA). The targeted suppression of key inflammatory pathways involved in joint inflammation and destruction allows better disease control, which, however, comes at the price of an elevated infection risk due to relative immunosuppression. The disease-related infection risk and the infection risk associated with the use of TNF-α inhibitors (infliximab, adalimumab, etanercept, golimumab and certolizumab pegol), rituximab, abatacept and tocilizumab are discussed. Risk factors clinicians need to take into account when selecting the most appropriate biologic therapy for RA patients, as well as precautions and screening concerning a number of specific infections, such as tuberculosis, intracellular bacterial infections, reactivation of chronic viral infections and HIV are reviewed. read more read less

Topics:

Infliximab (61%)61% related to the paper, Adalimumab (61%)61% related to the paper, Certolizumab pegol (60%)60% related to the paper, Abatacept (57%)57% related to the paper, Etanercept (57%)57% related to the paper
105 Citations
Journal Article DOI: 10.2174/157339708785133523
Animal Models of Osteoarthritis

Abstract:

The complex pathobiologic changes of human joint disease, particularly osteoarthritis (OA), normally take sev- eral decades to develop and may be influenced by a multitude of genetic and environmental factors. The need to clarify the molecular events that occur in joint tissues at the onset and during the progression of OA ha... The complex pathobiologic changes of human joint disease, particularly osteoarthritis (OA), normally take sev- eral decades to develop and may be influenced by a multitude of genetic and environmental factors. The need to clarify the molecular events that occur in joint tissues at the onset and during the progression of OA has necessitated the use of models, which, although imperfect, can exhibit many of the pathologic features that characterize the human disease. In vi- tro studies have proven invaluable in defining specific molecular and cellular events in degradation of joint tissues such as cartilage. However, to fully understand the complex inter-relationship between the different disease mechanisms, joint tis- sues and body systems, studying OA in animal models is necessary. Models of inflammatory arthropathies have proven predictive of clinical efficacy, with therapies that are beneficial in animals having significant benefit in treatment of rheumatoid arthritis in humans. While none of the available animal models of OA can truly be said to be predictive, as no anti-OA therapies have yet proven to be disease modifying in human trials, this approach represents a cornerstone for dis- covery of new anti-OA therapeutic targets and drugs. In this paper the available species and models of OA are reviewed and their potential utility discussed. read more read less
90 Citations
open accessOpen access Journal Article DOI: 10.2174/1573397111666150619095330
Psychosocial factors and central sensitivity syndromes
Leah M. Adams1, Dennis C. Turk2

Abstract:

Central sensitivity syndromes (CSSs) represent a heterogeneous group of disorders (e.g., fibromyalgia [FM], irritable bowel syndrome [IBS], chronic headache, temporomandibular disorders [TMDs], pelvic pain syndromes) that share many common symptoms, with persistent pain being the most prominent feature. Although the etiology ... Central sensitivity syndromes (CSSs) represent a heterogeneous group of disorders (e.g., fibromyalgia [FM], irritable bowel syndrome [IBS], chronic headache, temporomandibular disorders [TMDs], pelvic pain syndromes) that share many common symptoms, with persistent pain being the most prominent feature. Although the etiology and pathophysiology of CSSs are currently incompletely understood, central sensitization has emerged as one of the significant mechanisms. Given that there are currently no known cures for CSSs, people living with these disorders must learn to cope with and manage their symptoms throughout their lives. Medical interventions alone have not proven to be sufficient for helping people with CSSs manage their symptoms. A biopsychosocial perspective that considers the ways that biological, psychological, and social factors work independently and jointly to affect a person's experience is the most effective conceptualization and guide for effective treatment. In this article, we discuss several psychological and social features that may influence the experience of a person with CSS and their symptom management, regardless of their specific diagnosis. We highlight the longitudinal aspect of adjustment to illness, the distinction between psychosocial factors as causes of symptoms versus modifiers and perpetuators of symptoms, dispel the notion that all patients with the same diagnosis are a homogeneous group (the "patient-uniformity myth"), and acknowledge the importance of environmental and situational context on symptom management for individuals with any CSS. read more read less

Topics:

Psychosocial (55%)55% related to the paper, Biopsychosocial model (53%)53% related to the paper, Psychological intervention (51%)51% related to the paper
89 Citations
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Current Rheumatology Reviews format uses Vancouver citation style.

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Sure. We support all the top citation styles like APA style, MLA style, Vancouver style, Harvard style, Chicago style, etc. For example, in case of this journal, when you write your paper and hit autoformat, it will automatically update your article as per the Current Rheumatology Reviews citation style.

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To be honest, the answer is NO. The impact factor is one of the many elements that determine the quality of a journal. Few of those factors the review board, rejection rates, frequency of inclusion in indexes, Eigenfactor, etc. You must assess all the factors and then take the final call.

SHERPA/RoMEO Database

We have extracted this data from Sherpa Romeo to help our researchers understand the access level of this journal. The following table indicates the level of access a journal has as per Sherpa Romeo Archiving Policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

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S. No. Citation Style Type
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4. Author Year (Cited Pages)
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