Example of Gut Pathogens format
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Example of Gut Pathogens format Example of Gut Pathogens format Example of Gut Pathogens format Example of Gut Pathogens format Example of Gut Pathogens format Example of Gut Pathogens format Example of Gut Pathogens format Example of Gut Pathogens format Example of Gut Pathogens format Example of Gut Pathogens format Example of Gut Pathogens format Example of Gut Pathogens format Example of Gut Pathogens format Example of Gut Pathogens format Example of Gut Pathogens format Example of Gut Pathogens format Example of Gut Pathogens format Example of Gut Pathogens format
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Example of Gut Pathogens format Example of Gut Pathogens format Example of Gut Pathogens format Example of Gut Pathogens format Example of Gut Pathogens format Example of Gut Pathogens format Example of Gut Pathogens format Example of Gut Pathogens format Example of Gut Pathogens format Example of Gut Pathogens format Example of Gut Pathogens format Example of Gut Pathogens format Example of Gut Pathogens format Example of Gut Pathogens format Example of Gut Pathogens format Example of Gut Pathogens format Example of Gut Pathogens format Example of Gut Pathogens format
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open access Open Access

Gut Pathogens — Template for authors

Publisher: Springer
Categories Rank Trend in last 3 yrs
Parasitology #9 of 65 up up by 17 ranks
Gastroenterology #29 of 136 up up by 20 ranks
Infectious Diseases #64 of 288 up up by 36 ranks
Microbiology #45 of 150 up up by 19 ranks
Virology #24 of 69 up up by 7 ranks
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 239 Published Papers | 1445 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 03/07/2020
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Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

3.274

3% from 2018

Impact factor for Gut Pathogens from 2016 - 2019
Year Value
2019 3.274
2018 3.169
2017 2.809
2016 2.756
graph view Graph view
table view Table view

6.0

11% from 2019

CiteRatio for Gut Pathogens from 2016 - 2020
Year Value
2020 6.0
2019 5.4
2018 4.4
2017 4.0
2016 4.6
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 3% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 11% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

1.026

8% from 2019

SJR for Gut Pathogens from 2016 - 2020
Year Value
2020 1.026
2019 1.116
2018 1.183
2017 1.066
2016 1.317
graph view Graph view
table view Table view

1.34

24% from 2019

SNIP for Gut Pathogens from 2016 - 2020
Year Value
2020 1.34
2019 1.085
2018 1.007
2017 0.991
2016 1.104
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has decreased by 8% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 24% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Gut Pathogens

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Springer

Gut Pathogens

Approved by publishing and review experts on SciSpace, this template is built as per for Gut Pathogens formatting guidelines as mentioned in Springer author instructions. The current version was created on and has been used by 236 authors to write and format their manuscripts to this journal.

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Last updated on
03 Jul 2020
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ISSN
1606-8610
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Open Access
Yes
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Sherpa RoMEO Archiving Policy
White faq
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Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Citation Type
Numbered
[25]
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Bibliography Example
Blonder, G.E., Tinkham, M., Klapwijk, T.M.: Transition from metallic to tunneling regimes in superconducting microconstrictions: Excess current, charge imbalance, and supercurrent conversion. Phys. Rev. B 25(7), 4515–4532 (1982)

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1186/1757-4749-5-23
Commensal Clostridia: leading players in the maintenance of gut homeostasis
13 Aug 2013 - Gut Pathogens

Abstract:

The gastrointestinal tract is a complex and dynamic network where an intricate and mutualistic symbiosis modulates the relationship between the host and the microbiota in order to establish and ensure gut homeostasis. Commensal Clostridia consist of gram-positive, rod-shaped bacteria in the phylum Firmicutes and make up a sub... The gastrointestinal tract is a complex and dynamic network where an intricate and mutualistic symbiosis modulates the relationship between the host and the microbiota in order to establish and ensure gut homeostasis. Commensal Clostridia consist of gram-positive, rod-shaped bacteria in the phylum Firmicutes and make up a substantial part of the total bacteria in the gut microbiota. They start to colonize the intestine of breastfed infants during the first month of life and populate a specific region in the intestinal mucosa in close relationship with intestinal cells. This position allows them to participate as crucial factors in modulating physiologic, metabolic and immune processes in the gut during the entire lifespan, by interacting with the other resident microbe populations, but also by providing specific and essential functions. This review focus on what is currently known regarding the role of commensal Clostridia in the maintenance of overall gut function, as well as touch on their potential contribution in the unfavorable alteration of microbiota composition (dysbiosis) that has been implicated in several gastrointestinal disorders. Commensal Clostridia are strongly involved in the maintenance of overall gut function. This leads to important translational implications in regard to the prevention and treatment of dysbiosis, to drug efficacy and toxicity, and to the development of therapies that may modulate the composition of the microflora, capitalizing on the key role of commensal Clostridia, with the end goal of promoting gut health. read more read less

Topics:

Gut flora (61%)61% related to the paper, Dysbiosis (59%)59% related to the paper, Intestinal mucosa (53%)53% related to the paper
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597 Citations
open accessOpen access Journal Article DOI: 10.1186/1757-4749-1-6
A randomized, double-blind, placebo-controlled pilot study of a probiotic in emotional symptoms of chronic fatigue syndrome
19 Mar 2009 - Gut Pathogens

Abstract:

Chronic fatigue syndrome (CFS) is complex illness of unknown etiology. Among the broad range of symptoms, many patients report disturbances in the emotional realm, the most frequent of which is anxiety. Research shows that patients with CFS and other so-called functional somatic disorders have alterations in the intestinal mi... Chronic fatigue syndrome (CFS) is complex illness of unknown etiology. Among the broad range of symptoms, many patients report disturbances in the emotional realm, the most frequent of which is anxiety. Research shows that patients with CFS and other so-called functional somatic disorders have alterations in the intestinal microbial flora. Emerging studies have suggested that pathogenic and non-pathogenic gut bacteria might influence mood-related symptoms and even behavior in animals and humans. In this pilot study, 39 CFS patients were randomized to receive either 24 billion colony forming units of Lactobacillus casei strain Shirota (LcS) or a placebo daily for two months. Patients provided stool samples and completed the Beck Depression and Beck Anxiety Inventories before and after the intervention. We found a significant rise in both Lactobacillus and Bifidobacteria in those taking the LcS, and there was also a significant decrease in anxiety symptoms among those taking the probiotic vs controls (p = 0.01). These results lend further support to the presence of a gut-brain interface, one that may be mediated by microbes that reside or pass through the intestinal tract. read more read less

Topics:

Chronic fatigue syndrome (54%)54% related to the paper, Anxiety (53%)53% related to the paper, Placebo (51%)51% related to the paper
View PDF
470 Citations
open accessOpen access Journal Article DOI: 10.1186/S13099-016-0103-7
Inherent bacterial DNA contamination of extraction and sequencing reagents may affect interpretation of microbiota in low bacterial biomass samples
Angela Glassing1, Scot E. Dowd, Susan Galandiuk2, Brian R. Davis3, Rodrick J. Chiodini1
26 May 2016 - Gut Pathogens

Abstract:

The advent and use of highly sensitive molecular biology techniques to explore the microbiota and microbiome in environmental and tissue samples have detected the presence of contaminating microbial DNA within reagents. These microbial DNA contaminants may distort taxonomic distributions and relative frequencies in microbial ... The advent and use of highly sensitive molecular biology techniques to explore the microbiota and microbiome in environmental and tissue samples have detected the presence of contaminating microbial DNA within reagents. These microbial DNA contaminants may distort taxonomic distributions and relative frequencies in microbial datasets, as well as contribute to erroneous interpretations and identifications. We herein report on the occurrence of bacterial DNA contamination within commonly used DNA extraction kits and PCR reagents and the effect of these contaminates on data interpretation. When compared to previous reports, we identified an additional 88 bacterial genera as potential contaminants of molecular biology grade reagents, bringing the total number of known contaminating microbes to 181 genera. Many of the contaminants detected are considered normal inhabitants of the human gastrointestinal tract and the environment and are often indistinguishable from those genuinely present in the sample. Laboratories working on bacterial populations need to define contaminants present in all extraction kits and reagents used in the processing of DNA. Any unusual and/or unexpected findings need to be viewed as possible contamination as opposed to unique findings. read more read less

Topics:

Microbial DNA (58%)58% related to the paper, DNA extraction (52%)52% related to the paper, Metagenomics (51%)51% related to the paper
View PDF
380 Citations
open accessOpen access Journal Article DOI: 10.1186/S13099-019-0290-0
Virulence factors, prevalence and potential transmission of extraintestinal pathogenic Escherichia coli isolated from different sources: recent reports
21 Feb 2019 - Gut Pathogens

Abstract:

Extraintestinal pathogenic E. coli (ExPEC) are facultative pathogens that are part of the normal human intestinal flora. The ExPEC group includes uropathogenic E. coli (UPEC), neonatal meningitis E. coli (NMEC), sepsis-associated E. coli (SEPEC), and avian pathogenic E. coli (APEC). Virulence factors (VF) related to the patho... Extraintestinal pathogenic E. coli (ExPEC) are facultative pathogens that are part of the normal human intestinal flora. The ExPEC group includes uropathogenic E. coli (UPEC), neonatal meningitis E. coli (NMEC), sepsis-associated E. coli (SEPEC), and avian pathogenic E. coli (APEC). Virulence factors (VF) related to the pathogenicity of ExPEC are numerous and have a wide range of activities, from those related to bacteria colonization to those related to virulence, including adhesins, toxins, iron acquisition factors, lipopolysaccharides, polysaccharide capsules, and invasins, which are usually encoded on pathogenicity islands (PAIs), plasmids and other mobile genetic elements. Mechanisms underlying the dynamics of ExPEC transmission and the selection of virulent clones are still poorly understood and require further research. The time shift between colonization of ExPEC and the development of infection remains problematic in the context of establishing the relation between consumption of contaminated food and the appearance of first disease symptoms. What appears to be most difficult is to prove that ExPEC strains cause disease symptoms and to examine the mechanism of transition from the asymptomatic colonization of the intestines to the spreading of the bacteria outside the digestive system. A significant problem for researchers who are trying to ascribe ExPEC transmission to food, people or the environment is to draw the distinction between colonization of ExPEC and infection. Food safety is an important challenge for public health both at the production stage and in the course of its processing and distribution. Examination of the genetic similarity of ExPEC strains will allow to determine their origin from different sources. Many levels of genotyping have been proposed in which the typing of strains, plasmids and genes is compared in order to obtain a more complete picture of this complex problem. The aim of our study was to characterize E. coli strains isolated from humans, animals and food for the presence of bacterial genes encoding virulence factors such as toxins, and iron acquisition systems (siderophores) in the context of an increasing spread of ExPEC infections. read more read less

Topics:

Extraintestinal Pathogenic Escherichia coli (60%)60% related to the paper, Virulence (54%)54% related to the paper, Pathogenicity island (51%)51% related to the paper, Bacterial adhesin (50%)50% related to the paper
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357 Citations
open accessOpen access Journal Article DOI: 10.1186/1757-4749-5-10
Differences in gut microbiota composition between obese and lean children: a cross-sectional study
30 Apr 2013 - Gut Pathogens

Abstract:

An altered gut microbiota composition has recently been linked to obesity. The principal aim of this study is to investigate and compare the gut microbiota composition in obese and lean children. Secondly, associations between analysed gut bacterial species, dietary compounds, energy intake and biochemical blood parameters ar... An altered gut microbiota composition has recently been linked to obesity. The principal aim of this study is to investigate and compare the gut microbiota composition in obese and lean children. Secondly, associations between analysed gut bacterial species, dietary compounds, energy intake and biochemical blood parameters are evaluated. In this prospective cross-sectional study, 26 overweight/obese (mean BMI: 28.7 ± 6.5) and 27 lean (mean BMI: 16.5 ± 2.1) children aged 6 to 16 were included. Faecal samples were collected and subjected to selective plating and quantitative real-time PCR (qPCR) in order to determine the concentrations of bacterial species belonging to the genera: Bacteroides, Bifidobacterium, Clostridium, Staphylococcus and Lactobacillus. Matrix-assisted laser desorption/ionization time-of-flight mass spectrometry (MALDI-TOF MS) was applied for an in-depth identification of species of Bacteroides fragilis group. Differences in the concentrations of gut bacterial species between obese and lean children were statistically analysed using Mann Whitney U test. Subsequently, random forest analysis and multiple linear regression analysis were performed in order to test associations between gut bacterial species, dietary compounds and blood parameters. Obese children showed an elevated Firmicutes-to-Bacteroidetes ratio compared with lean children. Furthermore, low relative proportions of B. vulgatus and high concentrations of Lactobacillus spp. were observed in the obese microbiota. In all children, Staphylococcus spp. were positively associated with energy intake. Additionally, in obese children, Lactobacillus spp. were positively associated with plasma hs-CRP. Our findings corroborate a significant difference in the gut microbiota composition of important bacterial species between obese and lean children. In future, non-invasive manipulation of gut microbiota composition in early infancy could offer a new approach to manage childhood obesity and associated disorders. read more read less

Topics:

Gut flora (53%)53% related to the paper, Lactobacillus (51%)51% related to the paper
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342 Citations
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Frequently asked questions

1. Can I write Gut Pathogens in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Gut Pathogens guidelines and auto format it.

2. Do you follow the Gut Pathogens guidelines?

Yes, the template is compliant with the Gut Pathogens guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Gut Pathogens?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Gut Pathogens citation style.

4. Can I use the Gut Pathogens templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Gut Pathogens.

5. Can I use a manuscript in Gut Pathogens that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Gut Pathogens that you can download at the end.

6. How long does it usually take you to format my papers in Gut Pathogens?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Gut Pathogens.

7. Where can I find the template for the Gut Pathogens?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Gut Pathogens's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Gut Pathogens's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Gut Pathogens an online tool or is there a desktop version?

SciSpace's Gut Pathogens is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Gut Pathogens?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Gut Pathogens?”

11. What is the output that I would get after using Gut Pathogens?

After writing your paper autoformatting in Gut Pathogens, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Gut Pathogens's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Gut Pathogens?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Gut Pathogens. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Gut Pathogens?

The 5 most common citation types in order of usage for Gut Pathogens are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Gut Pathogens?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Gut Pathogens's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Gut Pathogens in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Gut Pathogens Endnote style according to Elsevier guidelines.

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