Showing papers in "Kidney International in 1987"

TL;DR: Morphologic evidence that cysts compressed adjacent renal parenchyma was observed in all kidneys and epithelial hyperplasia is a central element in the progressive enlargement of cysts.

TL;DR: The purpose of this editorial review is to analyze the physiologic significance of renal prostaglandin synthesis and the pathophysiologic consequences of inhibition of prostag landin production through the use of nonsteroidal anti-inflammatory drugs.

TL;DR: There was a significant increase in the number of interstitial T lymphocytes in all forms of proliferative glomerulonephritis when compared with the normal interstitial cell population.

TL;DR: The relative impact of age, sex, body build, hypertension, systemic atherosclerosis, intrarenal vascular disease and interstitial fibrosis on glomerulosclerosis and glomerular size was investigated using multiple linear regression.

TL;DR: These studies support the view that glomerular hypertension is an essential hemodynamic derangement responsible for progressive glomerul injury and reduction of capillary pressure can arrest the progression of remnant glomersular injury even when therapy is delayed until glomerULAR injury is established.

TL;DR: A 41-year-old man who reported vomiting, watery diarrhea, abdominal pain, and dark urine for about one week, and in the following few days, he also noted oliguria, a 10 kg weight gain, exertional dyspnea, severe headache, and visual disturbances.

TL;DR: Echocardiographic study of the left ventricle was performed in 57 selected, normotensive hemodialysis patients in comparison to 40 healthy controls matched for sex, age and blood pressure to find abnormalities in uremic patients and in dialysis patients.

TL;DR: Seven euvolemic Munich-Wistar rats were studied after acute CyA treatment to evaluate renal microcirculation and hormonal participation in this nephrotoxicity, observing an increase in afferent and efferent resistances and a decrease on glomerular plasma flow rate.

TL;DR: It is concluded that the ACE inhibitor lisinopril effectively reduces blood pressure and proteinuria in renal disease, and is probably also due to a fall in intraglomerular capillary pressure.

TL;DR: Intravenous administration of heterologous anti-rat thymocyte serum (ATS), which reacts with a Thy-1-like antigen present on rat glomerular mesangial cells, caused lytic, hypercellular, and sclerotic Mesangial lesions in Lewis rats.

TL;DR: In this paper, the role of IL 1 and IL 2 in the pathogenesis of immunological as well as inflammatory diseases was investigated. But, the authors did not consider the effect of cellulose membranes on PBMC cytokine production and the discrepancy between PBMC IL 1 production and serum IL 1 levels may be due to diminished excretion in patients with end-stage renal disease.

TL;DR: It is concluded that in CRF, chronic metabolic acidosis stimulates BCAA decarboxylation in skeletal muscle and this could contribute to the reduced intra- and extracellular concentrations of BCAA.

TL;DR: The finding of T lymphocytes as well as monocytes in glomeruli of proliferative nephritis suggests that cellular immune mechanisms may play a role in their pathogenesis, especially when crescents are present.

TL;DR: Observations suggest that in end-stage renal disease hypermagnesemia may retard the development of arterial calcifications in patients being treated by continuous ambulatory peritoneal dialysis.

TL;DR: The first demonstration of NEP having a major role in the catabolism of kinins is demonstrated, and the increase in UNaV and UV after phosphoramidon administration may be due to the inhibition of intrarenal kinin destruction.

TL;DR: Injection of the complex rapidly increased the urine volume of hypoalbuminemic patients who showed a marked resistance to this diuretic, and the resistance to furosemide in both NAR and hypo Albuminemic Patients may be explained on the same basis.

TL;DR: Findings demonstrate that components of the cell-mediated immune (CMI) system are present by MAb analysis, that subtypes cannot be differentiated by CMI constitution, and R to PM is related to intensity and composition of CMI involvement.

TL;DR: A 53-year-old man was referred to the Royal Melbourne Hospital for evaluation and treatment of episcleritis, a widower living in a country town and, apart from an appendectomy, had been in excellent health.

TL;DR: The exceptionally-high solute clearances obtained with continuous hemodialysis constitute a significant improvement in continuous renal replacement therapy.

TL;DR: Independent of any effect on systematic MAP, chronic administration of Ver protects against renal dysfunction, histological damage, nephrocalcinosis and myocardial calcification, and improves survival in the remnant model of chronic renal disease.

TL;DR: The data provide strong evidence for the existence of an AIDS-related glomerulopathy independent of i.v. drug use, and suggest that unrecognized co-factors may be important in the development of renal disease.

TL;DR: Seven patients with renal biopsy findings of mild glomerular abnormalities on light microscopy but with prominent accumulation of randomly-arranged fibrillar material in the mesangium and capillary walls on electron microscopy are described.

TL;DR: Abnormalities in lipid metabolism resulting from nephrotic syndrome may play a role in the progression of initial glomerular injury to focal and segmental glomerulosclerosis (FSGS), and both groups of rats were studied functionally and morphologically.

TL;DR: Antigens of the membrane attack complex of complement (MAC), such as C5, C6, C9 and MAC-related neoantigen(s), were demonstrated in the mesangium of 23 cases with IgA-glomerulonephritis and two cases with Henoch-Schönlein purpura nephritis by high specificity of the polyclonal antibodies.

TL;DR: A 65-year-old white man with long-standing ischemic cardiomyopathy and congestive heart failure was hospitalized because of 2 weeks of increasing dyspnea at rest, weight gain, and the onset of rapid atrial fibrillation.

TL;DR: One hundred fifty-four children aged eighteen years or younger from 83 families with autosomal-dominant polycystic kidney disease were studied by ultrasonography or excretory urography, finding that Renal area by ultr Masonography was greater among the ADPKD children compared to the SADPKD and NADPKD groups.