Journal ArticleDOI
Effects of O2 and CO2 on airway smooth muscle following pulmonary vascular occlusion.
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This article is published in Journal of Applied Physiology.The article was published on 1970-05-01. It has received 18 citations till now. The article focuses on the topics: Pulmonary compliance & Airway resistance.read more
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Journal ArticleDOI
Some remarks on the Gaussian beam summation method
TL;DR: In this paper, the authors used uniform asymptotic expansions to explain the behaviour of the Gaussian beam method and showed that the beam solution for head waves and in edge-diffracted shadow zones are both correct, but with governing parameters that are explicitly e-dependent.
Journal ArticleDOI
II. Effect of CO2 on afferent vagal endings in the canine lung.
TL;DR: Changes in slowly-adapting stretch receptor activity provide an adequate explanation for the pulmonary-CO2 ventilatory reflex, and the relationship between impulse frequency and lung PCO2 suggesting that these afferents may have a role in limiting CO2 loss under conditions causing hypocapnia, but be less effective in stimulating breathing during hypercapnia.
Journal ArticleDOI
Differentiation of Pulmonary Vascular from Parenchymal Diseases by Ventilation/Perfusion Scintiphotography
TL;DR: Defects observed in pulmonary perfusion scintiphotography often have been equated with a diagnosis of pulmonary embolism, but in fact, such defects have lacked diagnostic specificity because they lack diagnostic specificity.
Journal ArticleDOI
Effects of intrapulmonary CO2 and airway pressure on phrenic activity and pulmonary stretch receptor discharge in dogs
TL;DR: It appears that the reflex effects of both stimuli can be accounted by an effect on PSR activity, and the effects of lung stretch on ventilatory drive spans a wide range whereas intrapulmonary CO2 exerts an effect only at very low levels.
Journal ArticleDOI
The action of carbon dioxide on constricted airways.
TL;DR: In artificially ventilated open‐chest cats and dogs ventilation with 5–15% CO2 reversed the bronchoconstriction caused by drugs or by pulmonary artery occlusion.
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