Journal ArticleDOI
Fumonisin B1 exposure adversely affects porcine oocyte maturation in vitro by inducing mitochondrial dysfunction and oxidative stress.
Wenhui Li,Hongyu Zhao,Ruixue Zhuang,Yang Wang,Wei Cao,Yijing He,Yao Jiang,Rong Rui,Shiqiang Ju +8 more
TLDR
In this article, the effects and potential mechanisms of FB1 on mammalian oocytes during in-vitro maturation were explored using porcine oocyte complexes (COCs) as models.About:
This article is published in Theriogenology.The article was published on 2021-04-01. It has received 12 citations till now. The article focuses on the topics: Fumonisin B1 & Fumonisin.read more
Citations
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Journal ArticleDOI
Research Progress on Fumonisin B1 Contamination and Toxicity: A Review
TL;DR: In this article, a review of the current stage of FB1 contamination, its toxic effects of acute toxicity, immunotoxicity, organ toxicity, and reproductive toxicity on animals and humans are discussed.
Journal ArticleDOI
Grape Seed Proanthocyanidin Ameliorates FB1-Induced Meiotic Defects in Porcine Oocytes
TL;DR: In this paper, the authors explored whether Grape seed proanthocyanidin (GSP), a natural antioxidant, could alleviate the meiotic maturation defects of oocytes caused by fumonisin B1 exposure.
Journal ArticleDOI
Mito-Q promotes porcine oocytes maturation by maintaining mitochondrial thermogenesis via UCP2 downregulation.
Dan Zhou,Qingrui Zhuan,Yuwen Luo,Hongyu Liu,Lin Meng,Xingzhu Du,Guojuan Wu,Yunpeng Hou,Jun Liu,Xiangwei Fu +9 more
TL;DR: In this article , the effect of the mitochondria-targeted antioxidant Mito-Q on oocyte development and unravel the role of the antioxidant in mitochondrial ATP production and thermogenesis regulation.
Journal ArticleDOI
The natural occurrence, toxicity mechanisms and management strategies of Fumonisin B1：A review.
Zhicheng Gao,Kangxin Luo,Jinghui Peng,Qiuxiang Zhu,Chang Liu,Xiaoyue Wang,Shoujun Li,Haiying Zhang +7 more
TL;DR: Fumonisin B1 (FB1) contaminates various crops, causing huge losses to agriculture and livestock worldwide as discussed by the authors , which is particularly serious in developing countries, humid and hot regions.
Journal ArticleDOI
Biodegradation of Fumonisins by the Consecutive Action of a Fusion Enzyme
TL;DR: A safe and efficient fumonisin-degrading enzyme was discovered, which could be a new a technical method for hazard control of FBs in the future.
References
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Journal ArticleDOI
Production of fumonisin analogs by Fusarium species.
TL;DR: The fumonisins, a family of food-borne carcinogenic mycotoxins, were first isolated in 1988 from cultures of Fusarium verticillioides (Sacc.) Nirenberg and were shown to cause equine leukoencephalomalacia and the toxins produced by F. moniliforme were evaluated as “Group 2B carcinogens.”
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A review of the toxic effects and mechanisms of action of fumonisin B1.
TL;DR: Fumonisin B1 (FB1) is a mycotoxin produced by the fungus Fusarium verticillioides, which commonly infects corn and other agricultural products and has been classified as a possible carcinogen to humans.
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Impact of stress on oocyte quality and reproductive outcome.
TL;DR: Balance between level of ROS and antioxidants within the ovary are important for maintenance of female reproductive health and reduce ROS levels and protect against OS-mediated germ cell apoptosis and thereby depletion of germ cells from the Ovary.
Journal ArticleDOI
Analysis of Fusarium toxins in feed
TL;DR: An overview of the currently used methodology for the analysis of the Fusarium toxins fumonisins (FBs), moniliformin (MON), zearalenone (ZON) and type-A and -B trichothecenes in feeds, as well as other essential issues in mycotoxin analysis such as the availability of reliable calibrants and reference materials is given in this paper.
Journal ArticleDOI
Fumonisins and fumonisin analogs as inhibitors of ceramide synthase and inducers of apoptosis
Kena Desai,M. Cameron Sullards,Jeremy C. Allegood,Elaine Wang,Eva M. Schmelz,Michaela Hartl,Hans-Ulrich Humpf,Dennis C. Liotta,Qiong Peng,Alfred H. Merrill +9 more
TL;DR: It is found that fumonisins sometimes stimulate growth and inhibit apoptosis, possibly due to elevation of sphinganine 1-phosphate, which is known to have these cellular effects.