Open Access
GLOBOCAN 2012 v1.0, Cancer Incidence and Mortality Worldwide: IARC Cancer Base No. 11 [Internet]
J Ferlay,I Soerjomataram,M Ervik,R Dikshit,S Eser,Colin Mathers,M Rebelo,D.M. Parkin,David Forman,Freddie Bray,S Elser,M Ervick,Donald Maxwell Parkin +12 more
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The article was published on 2013-01-01 and is currently open access. It has received 2542 citations till now. The article focuses on the topics: Cancer.read more
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Does Metformin affect ER, PR, IGF-1R, β-catenin and PAX-2 expression in women with diabetes mellitus and endometrial cancer?
Anna Markowska,Monika Pawałowska,Violetta Filas,Konstanty Korski,Marian Gryboś,Stefan Sajdak,Anita Olejek,Wiesława Bednarek,Beata Śpiewankiewicz,Jolanta Lubin,Janina Markowska +10 more
TL;DR: There are still few studies confirming its favorable effect on endometrial cancer, and decreased ER expression in women with EC and DM2 receiving metformin needs further research to allow evaluation of its clinical significance.
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Caring for relatives with lung cancer in Europe: an evaluation of caregivers’ experience
TL;DR: Caregiving for lung cancer patients is associated with significant health/work impairments and costs, highlighting a need for increased, personalized caregiver support.
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DBGC: A Database of Human Gastric Cancer
TL;DR: The Database of Human Gastric Cancer (DBGC) is a comprehensive database that integrates various human Gastric cancer-related data resources that will greatly facilitate research regarding human gastric cancer in many fields.
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Polymorphisms in NFKB1 and TLR4 and interaction with dietary and life style factors in relation to colorectal cancer in a Danish prospective case-cohort study
TL;DR: A Danish prospective case-cohort study of 1010 CRC cases and 1829 randomly selected participants from the Danish Diet, Cancer and Health cohort found no strong gene-environment interactions between the examined polymorphism and diet and life style factors in relation to CRC risk.
Stromal cells of mesenchymal origin in breast cancer
TL;DR: In the in vivo experiments, the mesenchymal stromal cells inhibited and the fibroblasts enhanced the growth of breast cancer tumors, and the effect was due to both soluble factors and direct cell-cell contact.