Journal ArticleDOI
Prevalence of periimplant disease in partially edentulous patients: a practice‐based cross‐sectional study
TLDR
Smoking and compliance are important risk factors for periimplant inflammations in partially edentulous patients in a private dental practice.Abstract:
Objectives: Evaluation of the prevalence rates of periimplant mucositis and periimplantitis in partially edentulous patients in a private dental practice.
Material and methods: The data of 89 patients were collected (52 female, 37 male, age at time of implant placement: 51.8±10.3 years). All patients had been treated with dental implants of the same type and fixed superstructures between January 1999 and June 2006 (observational period: 68.2±24.8 months).
Results: The patient-related prevalence rate of periimplant mucositis (probing depth ≥4 mm and bleeding on probing [BOP]) was over all 44.9%. The respective rates in non-smokers without periodontal history were 30.4% and in smokers with periodontal history 80%. The multiple logistic regression analysis identified a significant association of mucositis with the independent variable “smoker” (odds ratio [OR] 3.77; P=0.023). The patient-related prevalence rate of periimplantitis (probing depth ≥5 mm, BOP/pus, radiographic bone loss) was 11.2% (smokers with periodontal history: 53.3%, non-smokers: 2.8%). No periimplant disease was diagnosed in non-smoking patients without periodontal history and with a good compliance after treatment. Statistical analysis identified a significant association of periimplantitis with “smoker” (OR: 31.58; P<0.001) and “compliance” (OR: 0.09; P=0.011). Periodontal history in general showed no significant association with periimplantitis.
Conclusions: Smoking and compliance are important risk factors for periimplant inflammations in partially edentulous patients.
To cite this article:
Rinke S, Ohl S, Ziebolz D, Lange K, Eickholz P. Prevalence of periimplant disease in partially edentulous patients: a practice-based cross-sectional study.
Clin. Oral Impl. Res. 22, 2011; 826–833
doi: 10.1111/j.1600-0501.2010.02061.xread more
Citations
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Journal ArticleDOI
The epidemiology of peri-implantitis.
TL;DR: Smoking and a history of periodontitis have been associated with a higher prevalence of peri-implantitis and factors that should be considered are the disease definition, the differential diagnosis, the chosen thresholds for probing depths and bone loss, differences in treatment methods and aftercare of patients, and dissimilarities in the composition of study populations.
Journal ArticleDOI
The frequency of peri-implant diseases: a systematic review and meta-analysis.
TL;DR: Long-term maintenance care for high-risk groups is essential to reduce the risk of peri-implantitis andformed consent for patients receiving implant treatment must include the need for such maintenance therapy.
Journal ArticleDOI
Peri-implant mucositis and peri-implantitis: A current understanding of their diagnoses and clinical implications
Paul S. Rosen,Donald S. Clem,David L. Cochran,Stuart J. Froum,Bradley S. McAllister,Stefan Renvert,Hom-Lay Wang +6 more
TL;DR: The purpose of this paper is to review the current knowledge concerning peri-implant mucositis and peri -implantitis to aid clinicians in their diagnoses and prevention and recognize that new information will continue to emerge.
Journal ArticleDOI
Impact of Maintenance Therapy for the Prevention of Peri-implant Diseases: A Systematic Review and Meta-analysis.
Alberto Monje,Luisiana Aranda,Karla Tatiana Diaz,Marco Antonio Alarcón,Robert A. Bagramian,Hom-Lay Wang,Andrés Catena +6 more
TL;DR: It can be concluded that implant therapy must not be limited to the placement and restoration of dental implants but to the implementation of PIMT to potentially prevent biologic complications and hence to heighten the long-term success rate.
Journal ArticleDOI
Peri-implant mucositis.
TL;DR: Peri-implant mucositis is an inflammatory lesion of the soft tissues surrounding an endosseous implant in the absence of loss of supporting bone or continuing marginal bone loss and biofilm accumulation disrupts the host-microbe homeostasis at the implant-mucosa interface, resulting in aninflammatory lesion.
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