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Albert-László Barabási
Researcher at Northeastern University
Publications - 463
Citations - 217721
Albert-László Barabási is an academic researcher from Northeastern University. The author has contributed to research in topics: Complex network & Network science. The author has an hindex of 152, co-authored 438 publications receiving 200119 citations. Previous affiliations of Albert-László Barabási include Budapest University of Technology and Economics & Lawrence Livermore National Laboratory.
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Universality classes for interface growth with quenched disorder
TL;DR: Numerical evidence is presented that there are two distinct universality classes characterizing driven interface roughening in the presence of quenched disorder, based on the behavior of $\lambda$, the coefficient of the nonlinear term in the growth equation.
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Identifying and modeling the structural discontinuities of human interactions.
Sebastian Grauwin,Michael Szell,Stanislav Sobolevsky,Philipp Hövel,Filippo Simini,Maarten Vanhoof,Zbigniew Smoreda,Albert-László Barabási,Albert-László Barabási,Albert-László Barabási,Carlo Ratti +10 more
TL;DR: This work analyzes several country-wide networks of telephone calls and uncovers a systematic decrease of communication induced by borders which is identified as the missing variable in state-of-the-art models.
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Integrating personalized gene expression profiles into predictive disease-associated gene pools
Jörg Menche,Jörg Menche,Jörg Menche,Emre Guney,Emre Guney,Amitabh Sharma,Amitabh Sharma,Amitabh Sharma,Patrick Branigan,Matthew J. Loza,Frédéric Baribaud,Radu Dobrin,Albert-László Barabási +12 more
TL;DR: It is shown that despite the high heterogeneity of the individual perturbation profiles, patients with asthma, Parkinson and Huntington’s disease share a broadpool of sporadically disease-associated genes, and that individuals with statistically significant overlap with this pool have a 80–100% chance of being diagnosed with the disease.
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Equilibrium phase diagrams for dislocation free self-assembled quantum dots
TL;DR: In this paper, it was shown that despite the large number of material constants entering the free energy of strained islands, there are only four topologically different phase diagrams describing the SAQD formation process.
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Comparison of an expanded ataxia interactome with patient medical records reveals a relationship between macular degeneration and ataxia
Juliette J. Kahle,Natali Gulbahce,Natali Gulbahce,Chad A. Shaw,Janghoo Lim,David E. Hill,Albert-László Barabási,Albert-László Barabási,Huda Y. Zoghbi +8 more
TL;DR: Novel protein interactions are identified for CACNA1A and ATXN7 linking them to other ataxia-causing proteins and theAtaxia network, and potential pathways that can contribute to the pathophysiology of ataxIA, MD, and diseases comorbid with ataxian diseases are suggested.