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Anton Reiner

Researcher at University of Tennessee Health Science Center

Publications -  163
Citations -  8850

Anton Reiner is an academic researcher from University of Tennessee Health Science Center. The author has contributed to research in topics: Striatum & Basal ganglia. The author has an hindex of 55, co-authored 163 publications receiving 8235 citations.

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Vascular endothelial growth factor increases release of gelatinase A and decreases release of tissue inhibitor of metalloproteinases by microvascular endothelial cells in vitro.

TL;DR: The data suggest that VEGF may modulate endothelial cell-derived MMP activity by increasing the abundance of gelatinase A; disinhibiting gelatinases A by decreasing the abundanceof TIMP-2; and disinhibititing preexisting collagenase by reducing levels of TIMP -1.
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Differential morphology of pyramidal tract-type and intratelencephalically projecting-type corticostriatal neurons and their intrastriatal terminals in rats

TL;DR: The clear differences between these two corticostriatal neuron types in perikaryal size and laminar location in the cortex, and in the size and shape of their intrastriatal terminals, suggest that they may differ in the nature of their influence on the striatum.
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Differential loss of striatal projection systems in Huntington's disease: a quantitative immunohistochemical study.

TL;DR: The notion that the striatal neurons preferentially projecting to the internal pallidal segment are, in fact, less vulnerable in HD than are the other striatal projection neuron types is supported.
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Organization and evolution of the avian forebrain.

TL;DR: The modern view of avian forebrain organization recognizes that the avian basal ganglia occupies no more of the telencephalon than is typically the case in mammals, and that it plays a role in motor control and motor learning as in mammals.
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Corticostriatal projection neurons - dichotomous types and dichotomous functions.

TL;DR: Findings have implications for understanding how the direct and indirect pathways carry out their respective roles in movement facilitation and movement suppression, and for understanding the role of corticostriatal synaptic plasticity in adaptive motor control by the basal ganglia.